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Hell mimimi


Of course, the love of the young and their potential fathers is not fully explained by oxytocin. Strangely enough, there is a higher nervous activity here - for example, activation of the reward system of the conditioned-reflex type with just one look at the photo of the beloved, even without any physical contact.
Nevertheless, the ability to experience love - especially for the young - is closely related to the hormonal status of the woman. Caring for babies is so tedious that it would be impossible without emotional involvement, and therefore almost all hormones associated with the reproduction process in mammals, to a greater or lesser extent, have a psychoactive effect and increase the tendency to tenderness (and at the same time to aggression towards offenders A cute cub is two sides of the same coin). In experiments on rats, it was found that animals deficient in their own sex hormones (with ovaries removed) are rather indifferent to the youngsters, but using injections of estradiol, progesterone and prolactin they can cause both lactation and starting parental behavior in relation to the first rat pups .
It seems that the opposite is also true: upon contact with the young, the hormonal status changes. This effect is well studied for oxytocin (it turned out that this is the most fashionable attachment hormone and grants are particularly well distinguished for its study), but in principle the same can happen with all other hormones involved in parental behavior. Anthropologist Marina Butovskaya in her popular science book “The secrets of sex. A man and a woman in the mirror of evolution ”writes that a woman just needs to look at the child so that her progesterone level rises in her blood. Moreover, this effect is observed not only at the sight of a human baby, but also at the sight of a kitten, a puppy or even a teddy bear - any big-headed and big-eyed creature with short legs.
In men, progesterone in the body is much less than in women, so our emotion at the sight of a kitten is usually completely incomprehensible to them.
But women (as a rule) still love men. The set of hormones associated with this feeling is not fundamentally different from the hormones of love for children. For example, prolactin, known primarily in connection with breastfeeding, also serves as a marker of orgasm. Scottish psychologist Stuart Brody, whose research is devoted to the most diverse aspects of human sexual behavior, in 2006 published an article in which the relationship between prolactin and orgasm was studied in detail. He invited 19 men and 19 women to his laboratory, instructed half to masturbate, and half asked to engage in vaginal sex with an orgasm by both partners (most surprisingly, he notes that ten men and nine women masturbated, and ten women and nine men had sex; who at the same time was in what relations with each other, is not reported). After orgasm, he measured the level of prolactin and found that sexual contact leads to a significantly greater release of this hormone than masturbation. Moreover, in women, the concentration of prolactin after sex is twice as high as in men. Brodie believes that the release of prolactin - is the main reason for experiencing relaxation and satisfaction after sex. Perhaps it is the difference in the level of this hormone that leads to the fact that men after sex, as a rule, easily switch to something else, while women still want to lie in bed, purr and cuddle.
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Hell mimimi

  1. Vizer V.A. Lectures on therapy, 2011
    On the subject - almost completely cover the difficulties in the course of hospital therapy, issues of diagnosis, treatment, according to the presentation, concise and quite accessible. Allergic diseases of the lungs Diseases of the joints Reiter's disease Sjogren's disease Bronchial asthma Bronchoectatic disease Hypertensive heart disease Glomerulonephrasafasditis Esophageal hernia Destructive lung diseases
  2. ALLERGIC LUNG DISEASES
    In recent decades there has been a significant increase in the number. patients with allergic diseases of the bronchopulmonary apparatus. Allergic lung diseases include exogenous allergic alveolitis, pulmonary eosinophilia, medicinal
  3. Exogenous allergic alveolites
    Exogenous allergic alveolitis (synonym: hypersensitive pneumonitis, interstitial granulomatous alveolitis) is a group of diseases caused by intense and, rarely, prolonged inhalation of antigens of organic and inorganic dusts and are diffuse, unlike pulmonary eosinophilia, and alveolar and interstitial structures. The emergence of this group
  4. TREATMENT
    1. General measures aimed at separating the patient with the source of antigen: compliance with sanitary requirements at the workplace, technological improvement of industrial and agricultural production, rational employment of patients. 2. Drug treatment. In the acute stage - prednisone 1 mg / kg per day for 1-3 days, followed by a decrease in dose
  5. CHRONIC EOSINOPHIL PNEUMONIA
    It differs from Leffler syndrome by a longer (more than 4 weeks) and severe course, up to severe intoxication, fever, weight loss, the appearance of pleural effusion with a high content of eosinophils (Lehrer-Kindberg syndrome). A long course of pulmonary eosinophilia, as a rule, is the result of a short-term, thorough examination of the patient in order to identify its cause. In addition to the reasons
  6. PULMONARY EOSINOPHILIA WITH ASTMATIC SYNDROME
    This group of diseases can include bronchial asthma and diseases with a leading bronchostatic syndrome, which are based on other etiological factors. These diseases include: 1. Allergic bronchopulmonary aspergillosis. 2. Tropical pulmonary eosinophilia. 3. Pulmonary eosinophilia with systemic manifestations. 4. Hyper-eosinophilic
  7. LITERATURE
    1. Respiratory Diseases: A Guide for Physicians: In 4 volumes. Edited by N.R. Paleev. T.4. - M .: Medicine. - 1990. - pp. 22-39. 2. Silverstov V.P., Bakulin MP Allergic lesions of the lungs // Wed. med. - 1987. - №12. - P.117-122. 3. Exogenous Allergic Alveolitis, Ed. A.G. Khomenko, St.Muller, V.Schilling. - M.: Medicine, 1987. -
  8. BRONCHOECTATIC DISEASE
    Bronchiectasis is an acquired (in some cases congenital) disease characterized by a chronic suppurative process in the irreversibly modified (expanded, deformed) and functionally defective bronchi mainly of the lower lung. ETIOLOGY AND PATHOGENESIS. Bronchiectasis is congenital in 6% of cases, being a defect of fetal development, a consequence of
  9. CLASSIFICATION OF BRONCHOECTASES
    (A.I. Borokhov, N.R. Paleev, 1990) 1. By origin: 1.1. Primary (congenital cysts) bronchiectasis. 1.1.1. Solitary (solitary). 1.1.2. Multiple. l..l-Z. Cystic lung. 1.2. Secondary (acquired) bronchiectasis. 2. According to the form of the expansion of the bronchi: 2.1. Cylindrical. 2.2. Bagular. 2.3. Spindly. 2.4. Mixed. 3. By severity
  10. LITERATURE
    1. Diseases of the respiratory system. A guide for physicians ed. N.R. Paleev. - M .: Medicine, 1990. - TZ, T.4. 2. Okorokov A.N. Treatment of diseases of internal organs: Practical guidance: In Zt. TI - Min.Vysh., Belmedkniga, 1997. 3. Harrison, T.R. Internal illnesses. - M .: Medicine, T.7,
  11. DISEASE (SYNDROME) REUTERS
    Reiter's disease (Reiter's syndrome, Fissenzhe-Leroy syndrome, urethro-oculo-synovial syndrome) is an inflammatory process that develops in most cases in close chronological connection with infections of the urinary tract or intestines and manifested by the classical triad of urethritis, conjunctivitis, arthritis. Most often, young (20-40) men who have had urethritis are ill. Women, children and the elderly
  12. ILLNESS (SYNDROME) SHEGREN
    The combination of dry keratoconjunctivitis, xerostomia and chronic polyarthritis was so thoroughly described by the Swedish ophthalmologist Sjögren (Shegren, 1933) that soon attracted the attention of clinicians from various countries to this very peculiar clinical phenomenon, although isolated observations of such a triad or individual manifestations of secretory glandular insufficiency were described earlier. Behind
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