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Deficient anemia


-Anamnesis: eating disorders, digestive disorders
-sm anemia: pallor of the skin and mucous membranes, reduced hemoglobin and red blood cells
-hyperplastic sm: an increase in the liver and spleen, according to the severity of the disease
- hemolytic sm: jaundice, splenomegaly, increase of indirect bilirubin in the blood
- hemorrhagic s-m is expressed only in severe forms
-labor diagnostic criteria: when there is iron deficiency anemia, the level of serum iron capillaries is reduced
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Deficient anemia

  1. 76. B12 DEFICIENCY ANEMIA
    Pernicious anemia develops due to vitamin B12 deficiency (daily requirement of 1-5 μg). In most cases, combined with fundal gastritis and achlorhydria. Pernicious anemia is an autoimmune disease with the formation of AT to the parietal cells of the stomach or the internal factor of Castle, however, there are B2-deficient anemias of alimentary genesis. Pernicious anemia can be congenital
  2. “DEFICIENT” ANEMIA.
    B12 - folic acid deficiency anemia. Hyperchromic anemia. The presence of megaloblasts (giant erythrocytes containing the nucleus) and megalocytes (giant erythrocytes with completed enucleation), and gigantic with polysegmented neutrophil nuclei. Detection in macrocytes of Jolly Taurus - remnants of the nuclear substance and Kebot rings - remnants of the nuclear membrane. Iron deficiency anemia. Hypochromic
  3. B12 (FOLIEVO) -DEFICITIVE ANEMIA
    The classic form of B12-deficient anemia is the so-called malignant, or pernicious, Addison-Birmere anemia. In Addison-Birmere disease, a diverse clinical and hematological syndrome, which characterizes the state of endogenous B12-avitaminosis in “pure” form (Fig. 41), is fully realized. To endogenous B12-avitaminosis, we also include perinitic-anemic
  4. Vitamin B12- and folic deficiency anemias
    Vitamin B12- and folic-deficient anemias belong to the group of macrocytic anemias. In essence, they are two different anemias, and they are rarely found in the same person at the same time. These anemias have similar pathogenetic mechanisms. They are associated, ultimately, with a violation of the synthesis of DNA and RNA with a deficiency of vitamin B! 2, folic acid, and some rare hereditary fermentopathies.
  5. Deficient anemia in young children
    Questions for repetition: 1. Hemogram of healthy children of different ages. 2. Indicators of red blood sprout in healthy children (life expectancy, the size and shape of red blood cells, the calculation and value of the color index). Test questions: 1. Deficiency anemia in children. Classification. 2. Iron deficiency anemia (IDA): 2.1. predisposing factors, risk groups. 2.2. etiopathogenesis of IDA.
  6. Vitamin B6-deficient anemia
    Develop with deficiency or increased intake of vitamin B6. Vitamin B6 is found in large quantities in fresh vegetables, cereals, yeast, meat, egg yolks and other foods. Therefore, the true hypo-or vitamin B6 vitamin deficiency is very rare and mostly in infants. Causes of vitamin Ba deficiency are unilateral milk feeding, long-term intestinal
  7. B-12-folic deficiency anemia
    B-12-folic deficiency anemias are associated with impaired RNA and DNA synthesis. Previously, this anemia was named after the authors who described it — Addison in 1849 and Birmer in 1872. Prior to the discovery of hepatic therapy, this disease was fatal, with which the old term "pernicious anemia" is connected, which means fatal or malignant anemia. At the end of the last century there were works about
  8. Enterogenic folic-B12-deficient anemias
    Symptomatic folic-B12-deficient anemias, associated with chronic intestinal diseases (enteritis such as sprue and celiac disease), as well as with the functional shutdown of a more or less significant segment of the small intestine (resection, atresia, diverticulosis), are combined under this name. ANEMIA AT THE SPRU Sprue (spru, dutch sprew — foam ”synonyms: aphtae tropicae, psilolis
  9. Vitamin B12-deficient anemia
    Vitamin B12-deficient
  10. Acquired forms of vitamin B12-deficiency anemia
    There are much more often than hereditary. Causes of acquired vitamin B12-deficient anemias: I. Inadequate intake of vitamin B12. 1. Maternal vitamin B12 deficiency (vegetarianism, pernicious anemia, sprue) leading to vitamin Bt2 deficiency in breast milk — megaloblastic anemia in breastfed children develops at 7–24 months (sometimes at an earlier age). 2
  11. HYPERCHROME (FOLIEVO-B12-DEFICIENCY) MACROCYTIC ANEMIA OF PREGNANT WOMEN
    Hyperchromic macrocytic anemia of pregnant women in conditions of our country is relatively rare. Much more often this anemia occurs in women of subtropical and tropical countries, and often against the background of malaria, helminthic invasion or intestinal lesions. According to the observations of Layrisse, in Venezuela, macrocytic anemia of pregnant women is a relatively frequent complication
  12. ANEMIA DUE TO TOXIC EXPOSURE OF THE BONE MARROW (MYETHOXICAL ANEMIA)
    THYROORTHEUS ANEMIA Thyroid stimulation anemia is anemia that develops on the basis of hypothyroidism. Experimental work confirms the important role of thyroid hormone - thyroxin - in stimulating normal blood formation. Animals deprived of the thyroid gland, quickly anemiziruyutsya, the introduction of the same thyroid drugs to thyroidectomized animals cures them from the effects of thyroid-induced
  13. ANEMIA UNDER CONDITIONS OF FUNCTIONAL AGASTRY (AGASTRAL ANEMIA)
    In contrast to the actual agastric anemia associated with partial or complete anatomical removal of the stomach, we distinguish anemia that develops in conditions of functional agastria, when the anatomically preserved stomach partially or completely turns off from the physiological process of digestion and, in particular, loses the ability to assimilate the external antianemic factor - Vitamin B12.
  14. Anemia associated with impaired synthesis of DNA and RNA (megaloblastic anemia)
    Megaloblastic anemia unites a group of acquired and hereditary anemias, a common feature of which is the presence of megaloblasts in the bone marrow. Regardless of the etiology of the patients, hyperchromic anemia with characteristic changes in the erythrocyte morphology is revealed - oval red blood cells, large (up to 12-14 microns or more). There are red blood cells with basophilic cytoplasm, in many of
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