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Inflammation of the esophagus (esophagitis)

Esophagitis is an inflammation of the mucous membrane of the esophagus. There are acute and chronic esophagitis.

Acute esophagitis

The main reason for the development of acute esophagitis can be mechanical and chemical irritants. In infants, esophagitis can occur due to exposure to mucous membranes of hot food when eating from a bottle. In childhood infectious diseases, it appears with diphtheria, scarlet fever, and measles. It can occur with injuries of the esophagus during probing, with the use of inhalation anesthesia. In addition, esophagitis in children occurs with the systematic use of coarse, poorly chewed food, with acute trauma swallowed by the bone, with burns of the esophagus by accidentally drunk chemicals (acids, alkalis, iodine, etc.). The most common cause may be a hiatal hernia.

Clinic. According to the clinical course and morphological signs, esophagitis can be catarrhal, erosive, hemorrhagic, pseudomembranous and necrotic. Children can not clearly differentiate their sensations and most often complain of pain behind the sternum. In case of hemorrhagic esophagitis, there may be bloody vomiting. In cases of pseudomembranous esophagitis, fibrin films are found in the vomit. Complications of necrotic esophagitis can be severe bleeding, mediastinitis. An abscess and phlegmon of the esophagus may occur. In these cases, the disease is very severe and is accompanied by unbearable pain behind the sternum, dysphagia, fever, and symptoms of severe intoxication.

Diagnostics. The diagnosis is established on the basis of the anamnesis, the main symptoms of the disease, esophagoscopy and x-ray examination in the absence of contraindications.

Treatment. The main thing is to eliminate the damaging factor. With any esophagitis, a sparing diet, liquid food at room temperature is prescribed. For anesthetic effect, anesthesin is prescribed at an age dose 4 times a day.

Chronic esophagitis is diagnosed with diaphragmatic hernia, gastric ulcer.
It is rare in children.

The main cause of the disease is a weakening of the closure of the lower sphincter of the esophagus, resulting in gastroesophageal reflux - throwing the gastric contents into the esophagus.

Clinic. The main complaints are heartburn, a burning sensation behind the sternum, belching sour, regurgitation, while the gastric contents can get into the respiratory tract, which causes a painful cough. One of the complications with a long course is cicatricial changes in the esophagus. In these cases, difficulty in the passage of food, a lump in the throat, and swallowing appear.

Diagnostics. The diagnosis is made on the basis of anamnestic data and data from an endoscopic examination of the esophagus when ulcers are found, especially in the lower esophagus, after the healing of which scars form.

Treatment. In the case of a chronic form of esophagitis, a sparing diet is prescribed. Food is taken 5-6 times a day with the exception of spicy, salty dishes. Mineral water is prescribed (Essentuki, Smirnovskaya). Reflux of acidic contents is prevented by providing a raised headboard. Of the medications prescribed: maalox - immediately after a meal, the dosage is determined individually; gastal - for children from 6 to 12 years old in half the dose of adults, 1/2 tablet 4 times a day, and also to reduce gastric secretion: omeprazole preparations.

In order to normalize the work of the esophagus sphincter, cerucal (metoclopromide) is prescribed - not more than 0.1 mg per kg of body weight for children from 2 to 14 years old for 4-6 weeks.

If you suspect an abscess and phlegmon, the patient is hospitalized in the surgical department. In these cases, the prognosis is poor.

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Inflammation of the esophagus (esophagitis)

  1. INFLAMMATION: DEFINITION, ESSENCE, BIOLOGICAL SIGNIFICANCE. MEDIATORS OF INFLAMMATION. LOCAL AND GENERAL MANIFESTATIONS OF INFLAMMATION. ACUTE INFLAMMATION: ETIOLOGY, PATHOGENESIS. MORPHOLOGICAL MANIFESTATION OF EXUSDATIVE INFLAMMATION. RESULTS OF ACUTE INFLAMMATION
    Inflammation is a biological, and at the same time a key, general pathological process, the appropriateness of which is determined by its protective and adaptive function aimed at eliminating the damaging agent and repairing damaged tissue. In medicine, to indicate inflammation, the term "um" is added to the name of the organ in which the inflammatory process develops - myocarditis, bronchitis,
  2. Esophagitis
    Esophagitis is an inflammation of the mucous membrane of the esophagus, most often caused by frequent returns of the contents of the stomach to the esophagus. See the Gullet (PROBLEMS) article, with the addition that a person suppresses anger and that the cause of this anger arises again and again, reinforcing it and preventing a person from swallowing some recent
  3. ESOFAGIT
    Esophagitis is one of the most common diseases of the esophagus. Men and women get sick equally often. Esophagitis is usually a secondary disease. They may be complicated by other diseases of the esophagus (achalasia of the cardia, malignant neoplasms) and the underlying parts of the gastrointestinal tract (peptic ulcer of the stomach and duodenum, cholecystitis, pancreatitis, etc.).
  4. Inflammation. Definition, essence, mediators of inflammation. Local and general manifestations of exudative inflammation, morphological manifestations of exudative inflammation. The answer is the acute phase. Ulcerative necrotic reactions with inflammation.
    1. The main processes that develop in the body in response to tissue damage are 1. amyloidosis 2. inflammation 3. regeneration 4. formation of granulomas 5. hyperplasia of cell ultrastructures 2. Inflammation is 1. hyperplasia of cell ultrastructures 2. restoration of lost structures 3 uncontrolled growth of cellular elements 4. exudative-proliferative response to damage 5. cellular
  5. Barrett's esophagus, adenocarcinoma of the esophagus
    There is no reliable data on the incidence of adenocarcinoma with Barrett's esophagus, but it is proved that the risk of the disease increases in this case by 20-40 times. Presumably, the mechanisms of carcinogenesis are similar to those in colon cancer - chronic damage to the epithelium and cell proliferation lead to genetic rearrangements and, ultimately, to the neoplastic process.
  6. Gastroesophageal reflux. Reflux esophagitis (ciphers K 20, K 21)
    Definition Reflux esophagitis is an inflammation of the mucous membrane of the distal esophagus, due to reflux of acidic gastric contents. Statistics. Gastroesophageal reflux episodically occurs in 10-14% of adults. Every tenth of this subpopulation of symptoms can be pronounced, which significantly impairs the quality of life. Etiology. Pathogenesis. Leading etiological
  7. Productive and chronic inflammation. Granulomatosis. The morphology of specific and non-specific inflammation.
    1. Chronic inflammation is manifested by a simultaneous combination of 1. failed repair 2. angiogenesis, scarring 3. reactive changes 4. tissue damage 5. embolism 2. Causes of chronic inflammation 1. acute infection 2. persistent infection 3. prolonged exposure to toxic substances 3. Chronic inflammation characterized by 1. deposition of amyloid 2. mononuclear infiltration
  8. The course of inflammation. Acute and chronic inflammation
    The course of inflammation is determined by the reactivity of the body, the type, strength, and duration of the phlogogen. There are acute, subacute and chronic inflammation. Acute inflammation is characterized by: - ​​intense course and relatively short (usually 1-2, maximum up to 4-6 weeks) duration (depending on the damaged organ or tissue, the degree and scale of their alteration,
  9. Inflammation Overview acute inflammation
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  10. Proliferation and outcomes of inflammation. Types of inflammation
    Inflammation always begins with damage and cell death. But at a certain stage, when the processes of repairing damage, cleansing of all the dead, alien to the body come into force, infiltration, suppuration and the processes of proteolysis and necrosis associated with them are stopped and recovery processes come to the fore. In accordance with this, the cellular composition of the inflammatory
  11. Nutrition for exacerbation of liver inflammation and acute gallbladder inflammation
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