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Congenital lobar emphysema

Congenital lobar emphysema is characterized by stretching of the parenchyma of the lobe (less often the segment) due to partial obstruction of the draining bronchus.

Hypothesis of pathogenesis:

1) underdevelopment or absence of cartilage of the bronchus;

2) hypertrophy of the mucous membrane of the bronchi with the formation of folds, mucous plugs;

3) compression of the bronchus from the outside by bronchogenic cysts, abnormally located vessels (favorite localization -

upper lobe of the left lung).

With x-ray and tomography, increased transparency of the affected part of the lung. Pulmonary pattern in this area is depleted or not traced at all. The diaphragm is flattened, its tour is limited. The mediastinum is displaced towards the unaffected lung.

Bronchological examination is uninformative, and most importantly, unsafe for patients, as it can lead to rupture of the emphysema inflated part of the lung.

Treatment. Surgical removal of the affected part of the lung.

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Congenital lobar emphysema

  1. Congenital lobar emphysema
    Pathophysiology Congenital lobar emphysema is characterized by over-inflation of one or more lobes of the lung. The most common cause is dysplasia of the bronchial cartilage, as a result of which on the exhale there is a collapse of large bronchi. The air that accumulates in the lungs acts like pneumothorax: the emphysematous lobe compresses the adjacent lobes, displaces the mediastinum and violates the venous
  2. Emphysema
    Pulmonary emphysema is a disease caused by widening of the airways in the deepest part of the bronchi. Pulmonary emphysema is usually a consequence of chronic bronchitis. With this disease, it is difficult for a person to breathe, as if he does not have enough air all the time. See the article BRONCHITIS, with the addition that pulmonary emphysema is a more important message from the body and requires immediate
  3. Emphysema
    Emphysema is characterized by excessive and steady expansion of the air and respiratory structures (or spaces) located distal to the terminal bronchioles, and the destruction of the walls of these structures without subsequent fibrosis. The expansion of the airways and respiratory spaces without destruction is the excess filling of the lungs with air (such an expansion of the end bronchi and alveoli is noted in
  4. Interstitial Pulmonary Emphysema
    Air leak syndrome is a group of pathological conditions characterized by accumulation of gas outside the alveolar space. Most often, a violation of the integrity of the alveoli occurs as a result of damage to the respiratory epithelium of the alveoli and terminal airways by high intrapulmonary pressure. Air leak syndrome combines interstitial pulmonary emphysema, pneumothorax,
    Emphysema - excessive airiness and an increase in lung volume due to damage or violation of the elasticity of the alveoli. Acute emphysema can develop after excessive physical exertion, it is also observed during an attack of bronchial asthma. Chronic emphysema occurs as a consequence of chronic bronchitis. Symptoms: rapid and intense breathing, shortness of breath with lowering of the sides,
  6. Increased airiness of the lungs (emphysema)
    Emphysema is a pathological expansion of the air spaces distal to the terminal bronchioles of compensatory, atrophic and obstructive genesis, accompanied by destructive changes in the alveolar walls. Emphysema is a form of chronic non-specific lung disease. It can be primary and secondary, which is a complication of bronchopulmonary
  7. Chronic bronchitis and emphysema
    Chronic bronchitis is a disease characterized by chronic or recurrent excessive secretion of mucus in the bronchi, leading to the appearance of a productive cough with annual exacerbations of up to 3 months or more in recent years. Emphysema is a disease caused by an increase in the air space of the end bronchioles as a result of destructive changes in their walls. Distinguish
  8. Chronic bronchitis and pulmonary emphysema
    Chronic bronchitis is a disease characterized by chronic or recurrent excessive secretion of mucus in the bronchi, leading to the appearance of a productive cough with annual exacerbations of up to 3 months or more in recent years. Emphysema is a disease caused by an increase in the air space of the end bronchioles as a result of destructive changes in their walls. Distinguish
    Roland G. Ingram, Jr. (Roland H. Ingram, Sr.) Chronic bronchitis and emphysema are two relatively independent diseases that often occur simultaneously and cause the development of chronic bronchial obstruction. The diagnosis of chronic bronchitis is established on the basis of history, the presence of bronchial obstructive syndrome, confirmed during functional studies
    .Emphysema of the lungs - damage to the lungs, characterized by a decrease in the elastic properties of the lung tissue, a violation of the structure of the alveolar walls, expansion of the air spaces of the lungs distal to the terminal bronchioles with the fall of the latter on the exhale and obstruction of the airways. In most cases, panacinar pulmonary emphysema develops. Ethiology - factors contributing to the stretching of the air
  11. Bacterial and viral airborne infections: influenza, parainfluenza, adenovirus infection, respiratory syncytial infection. Bacterial bronchopneumonia, lobar pneumonia.
    1. To supplement: Pulmonary atelectasis is _______________________. 2. The clinical and morphological form of bacterial pneumonia is determined by 1. type of inflammation 3. etiological agent 2. territory of the lesion 4. response of the body 3. In case of croupous pneumonia the consistency of the affected lobe 1. dense 2. flabby 3. not changed 4. The ability of the virus to selectively infect cells and tissues
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  13. Congenital Pneumonia
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  14. Congenital diseases
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  15. Congenital ichthyosis
    SYNONYMS Fetal ichthyosis, “Harlequin fetus”, colloidal fetus. DEFINITION Congenital ichthyosis is a term that combines several similar diseases, such as congenital ichthyosis, congenital ichthyosis such as a colloidal child, Brock's non-bullous ichthyosiform erythroderma, and ichthyosiform erythroderma bullous. CODE ICD-R 83 Other changes in the external integument specific to the fetus and
  16. Congenital malformations
    About 2-3% of newborns have serious congenital malformations. Embryologically, such defects are classified into three main classes (Table 36-6): • birth defects due to incomplete morphogenesis; • birth defects as a result of repeated morphogenesis; • birth defects as a result of aberrant morphogenesis. Incomplete morphogenesis is the most common
  17. Congenital heart defects
    Congenital heart disease is a pathological condition characterized by abnormalities in the development of the heart and great vessels, resulting from a violation of embryogenesis during the 2-8 weeks of pregnancy under the influence of adverse external factors (viruses, toxic substances, etc.) and internal (products of altered metabolism etc.) environment against the background of hereditary (polygenic)
  18. Congenital malformations
    Physical blockage Congenital malformation is a congenital malformation of an organ or tissue. Emotional blocking It is very difficult to find the exact cause of a birth defect, as in most cases it should be sought in a previous life. Parents should not assume that they are to blame. If this defect is inherited, read the metaphysical explanation of hereditary diseases at the beginning.
  19. Congenital factors
    As already noted, part of our knowledge of speech is innate. However, there are conflicting opinions about what kind of knowledge and to what extent is innate. One of the questions is related to the richness of the language. If the innate knowledge is very rich or detailed, then the process of mastering the speech should be similar for different languages, even if the possibilities for learning are not the same in different languages.
  20. Congenital heart defects
    Questions for repetition: 1. Features of the blood circulation of the fetus. 2. The timing of the closure of the oval window, arterial and venous ducts. Test questions: 1. Classification of congenital heart defects. 2. Clinical manifestations and diagnosis of congenital heart defects occurring with depletion of the pulmonary circulation: 2.1. Fallot's disease. 2.2. transposition of the great vessels with pulmonary stenosis
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