Licensed books on medicine
|
<< Previous | | Next >> | |
| Bacterial and viral airborne infections: influenza, parainfluenza, adenovirus infection, respiratory syncytial infection. Bacterial bronchopneumonia, lobar pneumonia.
|
1. Supplement: Atelectasis of the lungs is _______________________. 2. The clinical and morphological form of bacterial pneumonia is determined 1. type of inflammation 3. etiological agent 2. lesion territory 4. response of the body 3. With croupous pneumonia, the consistency of the affected lobe 1. tight 2. flabby 3. not changed 4. The ability of a virus to selectively infect cells and tissues is called: 1. virulence 4. phagocytosis 2. pathogenicity 5. tropism 3. invasiveness 5. Causes of death in the toxic form of severe influenza 1. thromboembolism 2. lobar pneumonia 3. toxic pulmonary edema 4. acute renal failure 5. acute heart failure 6. hemorrhages in vital organs 6. The airways of the lungs include: 1. acinus 4. trachea 2. terminal bronchiole 5. bronchi 3. alveolar duct 6. pneumocytes 7. Forms of bacterial pneumonia 1. lobar 4. staphylococcal 2. viral 5. bronchopneumonia 3. pneumococcal 8. In the stage of gray hepatization of croupous pneumonia, the exudate consists of 1. edematous fluid and bacteria 3. white blood cells and fibrin 2. red blood cells and fibrin 4. granulation tissue 9. The influenza virus has a tropism for 1. neurons 4. alveolar epithelium 2. hepatocytes 5. vascular endothelium 3. cardiomyocytes 6. epithelium of bronchioles 10. Severe influenza with pulmonary complications due to 1. accession of a secondary infection 2. the development of croupous pneumonia 3. cerebral hemorrhage 4. the development of focal pneumonia 11. The respiratory part of the lungs include: 1. acinus 4. trachea 2. terminal bronchiole 5. bronchi 3. alveolar duct 6. pneumocytes 12. The basis for the development of focal pneumonia is 1. atelectasis 3. acute emphysema 2. acute bronchitis 4. acute pneumonitis 13. In the stage of red hepatization of croupous pneumonia, the exudate consists of 1. white blood cells and fibrin 3. red blood cells and fibrin 2. granulation tissue 4. edematous fluid and white blood cells 14. The cytopathic effect of influenza virus is microscopically manifested. 1. inflammation 4. necrosis of the epithelium of bronchioles and alveoli 2. tissue edema 5. dystrophy of the epithelium of bronchioles and alveoli 3. hemorrhages 15. In severe influenza with pulmonary complications develops 1. purulent endobronchitis 3. metaplasia of the bronchial epithelium 2. purulent mesobronchitis 4. purulent destructive panbronchitis 16. Resorption atelectasis occurs when: 1. foreign body aspiration 2. exudative pleurisy 3. deficiency in the synthesis of surfactant 4. interstitial fibrosis 5. chronic bronchitis 17. The most important complications of bronchopneumonia 1. abscess 4. carnification 2. pleurisy 5. pneumosclerosis 3. emphysema 6. chronic bronchitis 18. In the inflow stage of croupous pneumonia, the exudate consists of 1. white blood cells and fibrin 3. red blood cells and fibrin 2. granulation tissue 4. edematous fluid and bacteria 19. Joining a secondary infection with influenza is associated with the action of the virus 1. immunosuppressive 3. cytopathic 2. neuropathic 4. vasopathic 20. Metaphorically mild in severe influenza with pulmonary complications is called 1. "greasy" 3. "shock" 5. "big red" 2. "cellular" 4. "big white" 6. "big motley" 21. Compression atelectasis occurs when: 1. foreign body aspiration 4. interstitial fibrosis 2. exudative pleurisy 5. chronic bronchitis 3. deficiency in the synthesis of surfactant 22. Depending on the volume of the lesion with bronchopneumonia, foci are distinguished 1. acinous 4. polysegmental 2. shared 5. total 3. segmental 23. To establish compliance: VOLUME OF THE POINT OF DAMAGE OF A PNEUMONIA 1. share a) focal 2. acinus b) croupous 3. lobule 4. segment 5. multiple segments 24. The immunosuppressive effect of the virus is manifested 1. hemorrhages 2. autoimmunization 3. activation of neutrophil phagocytosis 4. suppression of phagocytosis of neutrophils 5. suppression of macrophage chemotaxis 25. The characteristic appearance of the lung in severe influenza with pulmonary complications is due to 1. inflammation 4. abscess formation 2. pneumosclerosis 5. foci of emphysema 3. hemorrhages 6. foci of atelectasis 26. Spotted atelectasis occurs when: 1. foreign body aspiration 2. exudative pleurisy 3. deficiency in the synthesis of surfactant 4. interstitial fibrosis 5. chronic bronchitis 27. A synonym for focal pneumonia 1. croupous 3. bronchopneumonia 2. pleuropneumonia 4. interstitial 28. Clinical sign - pleural friction noise - characteristic of pneumonia 1. focal 2. croupous 3. interstitial 29. To establish compliance: MORPHOLOGICAL CHARACTER AT INFLUENZA CHARACTER OF CHANGES 1. interstitial inflammation a) general 2. discirculatory disorders b) local 3. necrosis of the epithelium of the bronchi and alveoli 4. dystrophy of the parenchymal organs 5. dystrophy of the epithelium of the bronchi and alveoli 30. Brain changes in severe influenza presented 1. serous meningitis 4. hydrocephalus 2. neuronal degeneration 5. calcification 3. hemorrhages 31. Contraction atelectasis occurs when: 1. foreign body aspiration 2. exudative pleurisy 3. deficiency in the synthesis of surfactant 4. interstitial fibrosis 5. chronic bronchitis 32. Set the correct sequence: STAGES OF LARGE PNEUMONIA 1. red guardianship 2. gray ward 3. permissions 4. tide 33. For croupous pneumonia, the staging process 1. characteristic 2. not characteristic 34. General changes in the flu 1. dystrophy and necrosis of the epithelium of the bronchi and alveoli 2. dystrophy of the parenchymal organs 3. discirculatory disorders 4. interstitial inflammation 5. systemic vasculitis 35. In the toxic variant of severe influenza, changes are more pronounced 1. general 2. local 36. Supplement: The pathomorphogenesis of RDSV is associated with ____________________________. 37. To establish compliance: STAGE OF LARGE PNEUMONIA DURATION 1. tide a) 1st day 2. red guardianship b) 2 - 3 days 3. gray guardianship c) 5 - 7 days 4. Permissions d) 7-14 days d) 25 days f) 40 days 38. Atelectasis of the lung - a change in lung tissue - in the form 1. increase airiness 2. inflammation 3. collapse 4. necrosis 39. Forms of influenza according to the nature of the course 1. mild 4. moderate 2. severe 5. relapsing 3. abortive 40. Extrapulmonary complications of severe influenza 1. chronic sinusitis 3. arachnoiditis 2. pneumofibrosis 4. encephalitis 41. Supplement: Nosocomial pneumonia is _________________________. 42. To establish compliance: STAGE OF LARGE PNEUMONIA COMPOSITION OF EXUDATE 1. tide a) edematous fluid and bacteria 2. red hepatization b) eosinophils and neutrophils 3. gray hepatitis c) macrophages and plasmocytes g) red blood cells and fibrin d) white blood cells and fibrin 43. Adult distress syndrome is associated with: 1. pulmonary edema 2. surfactant deficiency 3. diffuse damage to the capillaries of the alveolar septum 44. With a mild form of influenza, inflammation develops in the mucous membrane of the upper respiratory tract 1. purulent 2. catarrhal 3. fibrinous 4. hemorrhagic 5. serous - hemorrhagic 45. Supplement: A distinctive feature of respiratory syncytial infection is the defeat of ________________________. 46. Supplement: Bronchopneumonia is __________________________________. 47. Supplement: Lobar pneumonia often causes ___________________________. 48. With croupous pneumonia, pleura develops on the pleura 1. serous 2. croupous 3. diphtheria 49. With a mild form of influenza in the mucous membrane of the upper respiratory tract, you can identify 1. anemia 2.
gland hypersecretion 3. vascular congestion 4. sclerosis of the submucosal layer 5. dystrophy and disquamation of the epithelium 6. lymphoid cell infiltration 50. The lumen of the alveoli during respiratory syncytial infection is filled: 1. serous exudate 2. fibrinous exudate 3. giant cells 4. hemorrhagic exudate 5. desquamated alveocytes 51. Determine the type of inflammation if bronchopneumonia is caused by pneumococcus: 1. serous-fibrinous inflammation, microbial edema along the periphery of the foci of inflammation 2. hemorrhagic inflammation with suppuration and necrosis of the walls of the bronchi and alveolar moves 3. serous-purulent inflammation involving the interstitium 52. Pulmonary complications of lobar pneumonia 1. lung gangrene 4. carnification 2. lung abscess 5. fibrinous pleurisy 3. bronchiectasis 6. pulmonary thromboembolism 53. In croupous pneumonia, the affected lobe 1. fleshy consistency, airless 2. testy consistency, aerial 3. macroscopically unchanged 54. With moderate flu, inflammation develops in 1. trachea 4. conjunctiva 2. pleura 5. upper respiratory tract 3. bronchial tubes 55. In the small bronchi and bronchioles with respiratory syncytial infection observed 1. catarrhal inflammation 2. swelling of the mucosa 3. damage to the epithelium 4. epithelial proliferation with the formation of symplasts 5. leukocyte infiltration 56. Determine the type of inflammation if bronchopneumonia is caused by staphylococcus: 1. serous-fibrinous inflammation, microbial edema along the periphery of the foci of inflammation 2. hemorrhagic inflammation with suppuration and necrosis of the walls of the bronchi and alveolar moves 3. serous-purulent inflammation involving the interstitium 57. In focal pneumonia, the process begins with 1. interstitial tissue 3. bronchi 2. tissue of the alveoli 4. pleura 58. The first stage of croupous pneumonia 1. red guardianship 3. permissions 2. gray hepatitis 4. tide 59. With moderate flu, inflammation develops in the trachea and bronchi 1. serous hemorrhagic 3. catarrhal 2. fibrinous 4. purulent 60. Perform compliance: MANIFESTATION VIRUS VIRUS 1. A 1. leads to sporadic cases 2. B 2. greatest epidemiological hazard 3. C 3. causes local outbreaks and epidemics 61. Determine the type of inflammation if bronchopneumonia is caused by streptococcus: 1. serous-fibrinous inflammation, microbial edema along the periphery of the foci of inflammation 2. hemorrhagic inflammation with suppuration and necrosis of the walls of the bronchi and alveolar moves 3. serous-purulent inflammation involving the interstitium 62. With lobar pneumonia, the process begins with 1. pleura 3. alveolar tissue 2. bronchi 4. interstitial tissue 63. The second stage of croupous pneumonia 1. tide 3. gray ward 2. permissions 4. red guardianship 64. Microscopic changes in the mucous membrane of the larynx and trachea in severe influenza with pulmonary complications are represented by inflammation 1. catarrhal 2. phlegmonous 3. granulomatous 4. with the formation of polyps 5. fibrinous hemorrhagic 65. The causative agent of adenovirus infection enters the body through: 1. intestinal epithelium 2. epithelium of the meninges 3. airway epithelium 4. skin conjunctival epithelium 66. To supplement: acute pneumonitis is ______________________________. 67. Pleural empyema is a common complication. 1. bronchiectasis 3. lung emphysema 2. focal pneumonia 4. pneumosclerosis 68. The third stage of croupous pneumonia 1. tide 3. gray ward 2. permissions 4. red guardianship 69. Variants of severe influenza 1. toxic 3. hepatotoxic 2. myocarditis 4. with pulmonary complications 70. For a mild form of adenovirus infection, the symptoms are: 1. catarrhal conjunctivitis 2. enlarged lymph nodes and spleen 3. purulent necrotic bronchitis 4. interstitial inflammation in the liver and kidneys 5. the presence of giant cells in the epithelium 71. Synonyms for croupous pneumonia include: 1. lobar 4. hypostatic 2. purulent 5. fibrinous 3. pleuropneumonia 72. The staged course of the process is characteristic of pneumonia. 1. focal 2. croupous 3. interstitial 73. In the classic course of croupous pneumonia 1. 4 stages are distinguished 3. 2 stages are distinguished 2. 3 stages are distinguished 4. no staging 74. The toxic variant of severe influenza is characterized by 1. hemorrhage 2. purulent inflammation 3. necrotic nephrosis 4. catarrhal inflammation 5. hemorrhagic pulmonary edema 75. The development of a severe form of adenovirus infection is associated with: 1. intoxication 2. Secondary reproduction of the virus in organs 3. generalization of the virus 4. early age 5. lower immunity 6.a secondary infection ANSWERS No. | the answers | No. | the answers | | A pathological process characterized by reversible incomplete expansion or collapse of a part or whole lung that previously contained air | 39. | 1, 2, 4 | | 2, 3, 4 | 40. | 3, 4 | | 1 | 41. | Pneumonia that developed no earlier than 48 hours after hospitalization | | 5 | 42. | 1a, 2g, 3d | | 3, 6 | 43. | 3 | | 2, 4, 5 | 44. | 2 | | fifteen | 45. | Distal airways | | 3 | 46. | Focal pneumonia, which is characterized by the development in the pulmonary parenchyma of foci of acute inflammation ranging in size from the acinus to the segment associated with the affected bronchiole | | 4, 5, 6 | 47. | Pneumococcus | | 14 | 48. | 2 | | 1, 3, 6 | 49. | 2, 3, 5, 6 | | 2 | fifty. | 1, 3, 5 | | 3 | 51. | 1 | | 4, 5 | 52. | 1, 2, 4 | | 4 | 53. | 1 | | fifteen | 54. | 1, 3, 5 | | 1, 2, 4, 5 | 55. | 3, 4 | | 4 | 56. | 2 | | 1 | 57. | 3 | | 6 | 58. | 4 | | 2 | 59. | 1 | | 1, 3, 4, 5 | 60. | A-2, B-3, C-1 | | 1b, 2a, 3a, 4a, 5a | 61. | 3 | | 2, 4, 5 | 62. | 3 | | 1, 3, 4, 5, 6 | 63. | 4 | | 3 | 64. | 5 | | 3 | 65. | 1, 3, 4 | | 2 | 66. | Interstitial pneumonia, which is characterized by the primary development of acute inflammation in the alveolar wall and pulmonary interstitium with possible secondary formation of exudate in the lumen of the alveoli. | | 1a, 2a, 3b, 4a, 5b | 67. | 2 | | 1, 2, 3 | 68. | 3 | | 4 | 69. | 14 | | 4 - 1 - 2 - 3 | 70. | 1, 2, 5 | | 1 | 71. | 1, 3, 5 | | 2, 3, 4 | 72. | 2 | | 1 | 73. | 1 | | Damage to the endothelium of capillaries and first-order pneumocytes in the area of the airborne barrier, followed by the development of respiratory failure | 74. | fifteen | | 1a, 2b, 3c, 4g | 75. | 2, 3, 6 | | 3 | | |
| << Previous | | Next >> | = Skip to textbook content = |
| Bacterial and viral airborne infections: influenza, parainfluenza, adenovirus infection, respiratory syncytial infection. Bacterial bronchopneumonia, lobar pneumonia.
| - Bacterial airborne and intestinal infections
Airborne infections include diphtheria, scarlet fever, and meningococcal infection. They have a number of common features: airborne transmission predominates; the entrance gate is the upper respiratory tract; all diseases are characterized by severe toxicosis; more often children are ill. The seasonality of the disease is characteristic (autumn winter period). Diphtheria - an acute infectious disease caused by
- Bacterial airborne infections
Diphtheria Diphtheria is an acute infectious disease characterized by fibrinous inflammation in the area of the entrance gate and general intoxication. Currently, there is an increase in the incidence of diphtheria (especially among adults). Etiology and pathogenesis. • The disease is caused by toxigenic, ie producing exotoxin, strains of Corynobacteriae diphteriae. • People without
- VIRAL AIR-DROP INFECTIONS
One of the most common respiratory diseases is airborne viral infections, which usually occur acutely and sometimes become epidemics. The source of infection is infected people. For these diseases, as well as for other infections, a combination of local and general changes is characteristic. The most common are flu, parainfluenza,
- Acute respiratory viral infection and flu
Symptoms: manifestations of rhinitis, nasopharyngitis, pharyngitis, tonsillitis. ¦ More severe - with toxicosis and febrile temperature for 3-6 days - the flu is leaking. ¦ Long-term (4-6 days) fever is characteristic of adenovirus infection. ¦ Bacterial complications (sinusitis, otitis media, pneumonia), if they occur, then more often in the 1-2 days of illness, and can be detected at the first examination. Treatment Patient
- AIR DROP INFECTIONS
Airborne infections are a group of acute inflammatory diseases with predominant localization of changes in the upper respiratory tract. Features of airborne infections. 1. Airborne transmission. 2. The variety of pathogens: viruses, bacteria, protozoa, fungi. 3. The entrance gate is the mucous membrane of the upper respiratory tract. 4.
- AIR DROP INFECTIONS
A number of human infectious diseases are transmitted primarily by airborne droplets. These include diphtheria, measles, scarlet fever, meningococcal infection and others. Since these diseases are predominantly found in childhood, they are often called "childhood infections." Diphtheria is an acute infectious disease caused by diphtheria bacillus, characterized mainly
- The most common childhood airborne infections
Flu. This is an airborne infection, the most intense infectious disease. Influenza periodically occurs in the form of large epidemics, during which 30 to 70% of the population is ill in large cities. The causative agent of influenza is a virus characterized by variability. There are four types of influenza virus: A, B, C and O. In recent years, varieties of virus A have been identified, which are called A1 and
- Physiotherapy exercises for children often suffering from acute respiratory viral infections, pneumonia
To improve the work of the respiratory system, increase lung ventilation, improve blood circulation in them, resolve the inflammatory foci, treatment with posture, massage, and special exercises are recommended. Treatment position. In the acute period of the disease with respiratory viral infections, a relatively frequent change in the position of the child is recommended. You have to turn it from side to side, on your back,
- Airborne infections
Airborne droplets
- Bacterial infections
Of these, focal pneumonia is of particular importance, not only as one of the most frequent infectious lesions of a person, but also as a banal, sometimes severe, complication of various underlying diseases and as a direct cause of death of many patients. Almost any type of microbe with a sufficient degree of virulence can cause focal pneumonia in certain cases that have this
- Intrauterine bacterial infection of the fetus
Intrauterine bacterial infection of the fetus can be caused by pathogenic (staphylococcus, streptococcus, etc.), and conditionally pathogenic (enterobacteria, Klebsiella, Proteus, etc.) microorganisms, as well as mycoplasmas. The causative agents of infection can penetrate the fetal body transplacentally in the presence of focal infection foci in the mother's body (tonsillitis, sinusitis, dental caries, pyelonephritis and
- Bacterial intestinal infections
• Are the most common intestinal infections. • Among etiological factors, the most important role is played by gram-negative microorganisms belonging to the enterobacteriaceae family: Escherichia, Salmonella, Shigella, Enterobacter, Proteus, Yersinia, etc. The pathogenesis of intestinal infections caused by gram-negative microorganisms is largely determined by the action of toxins. a. Enterotoxin
- Abstract. Бактериальные инфекции, передающиеся половым путём, 2009
ВВЕДЕНИЕ Бактериальные инфекции, передающиеся половым путём Сифилис Гонорея Хламидиоз Гарднереллез Методы индивидуальной профилактики болезней, передающихся половым путем Заключение. Список использованной
- Пренатальные бактериальные инфекции
Пренатальные бактериальные
- Respiratory syncytial viral infection
Respiratory syncytial viral infection is an acute respiratory disease characterized by mild intoxication and a predominant lesion of the lower respiratory tract. Respiratory syncytial viruses are currently considered the main cause of bronchiolitis and pneumonia in children under the age of 1 year, as well as one of the most common respiratory pathogens.
|
|