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| Bowel disease. Infectious enterocolitis (dysentery, typhoid fever, cholera). Nonspecific ulcerative colitis. Crohn's disease. Coronary bowel disease. Appendicitis. Colon cancer
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1. Macroscopic characteristics of the small intestine with cholera enteritis 1. gray-yellow film tightly welded to the wall 2. ulceration of the mucous membrane 3. multiple hemorrhages 4. wall sclerosis 2. Elements of the pathogenesis of typhoid fever 1. bacteremia 2. bacteriocholia 3. cerebral swelling 4. exudative inflammation 5. hypersensitivity reaction in the lymphoid apparatus 3. The modern concept of the pathogenesis of appendicitis 1. alimentary 2. metabolic 3. immunological 4. angioneurotic 5. infectious and allergic 4. The cause of coronary bowel disease may be 1. vein thrombosis 2. diverticulosis 3. arterial thrombosis 4. intestinal infarction 5. intestinal stricture 6. embolism of arterial systems 5. Distant intestinal cancer metastases are found in 1. lungs 4. adrenal glands 2. kidney 5. lymph nodes 3. liver 6. Intestinal complications of typhoid fever 1. peritonitis 3. bleeding 2. perforation 4. cicatricial stenosis 7. Extraintestinal complications of dysenteric colitis 1. sepsis 4. focal pneumonia 2. amyloidosis 5. croupous pneumonia 3. encephalopathy 6. pylephlebitic liver abscesses 8. Clinical and morphological forms of acute appendicitis 1. granulomatous 4. phlegmonous 2. recurrent 5. primary 3. superficial 6. simple 9. The color of a heart attack with coronary bowel disease 1. white 2. hemorrhagic 3. white with a hemorrhagic nimbus 10. Macroscopic variants of colorectal carcinomas 1. circular 4. in the form of “cauliflower” 2. mushroom-shaped 5. with hidden infiltrating growth 3. plaque-like 11. Forms of typhoid fever depending on the localization of local changes 1. ileotif 4. hepatotif 2. colotif 5. meningotif 3. gastrotyphoid 6. cholangiotif 12. Morphological characteristics of the second stage of dysenteric colitis 1. granulomatosis 2. deep necrosis 3. fibrinous film 4. a large amount of fibrin 5. diffuse leukocyte infiltration 13. Destructive forms of acute appendicitis 1. granulomatous 4. phlegmonous 2. apostematous 5. gangrenous 3. superficial 6. simple 14. In chronic intestinal ischemia can be detected 1. heart attacks 3. ulcerations 2. inflammation 4. hemorrhages 15. Microscopic variants of colon cancer 1. mucous 4. squamous 2. adenocarcinoma 5. oat 3. cricoid cell 16. Specific Complications of Cholera 1. splenomegaly 4. cholera meningitis 2. ulcerative gastritis 5. post-cholera uremia 3. cholera typhoid 17. The preferred route of transmission of bacterial dysentery 1. airborne 4. parenteral 2. fecal-oral 5. pin 3. transmission 18. Pilephlebitic liver abscesses in acute appendicitis are a consequence 1. thromboembolism from the appendicular artery 2. self-amputation of the appendix 3. inflammation of the veins of the appendix 4. perforation of the appendix 19. Intestinal complications of Crohn's disease 1. fistulas between the intestines 2. bowel obstruction 3. protein-free enteropathy 4. fibrous strictures 5. peptic ulcers 20. The pathway of contracting typhoid fever 1. airborne 4. parenteral 2. fecal-oral 5. pin 3. transmission 21. In typhoid fever, changes are localized in the elements of the intestinal wall 1. muscle layer 4. serous membrane 2. nerve plexuses 5. mucous membrane 3. Peyer's plaques 6. solitary follicles 22. Intestinal complications of dysenteric colitis 1. amyloidosis 4. abscess formation 2. perforation 5. cicatricial stenosis 3. bleeding 23. Morphological forms of acute appendicitis 1. erosive 3. hemorrhagic 2. gangrenous 4. phlegmonous and ulcerative 24. Idiopathic inflammatory bowel disease 1. Crohn's disease 4. fibrosing colitis 2. ulcerative colitis 5. necrotizing enterocolitis 3. Whipple's disease 25. Exophytic colorectal cancers often grow in 1. cecum 4. in the ascending region 2. transverse colon 5. rectum 3. sigmoid colon 26. Characteristics of enteritis in the first week of typhoid fever 1. granulomatous 4. catarrhal 2. diphtheria 5. croupous 3. phlegmonous 6. ulcerative 27. Type of inflammation in the second stage of dysenteric colitis 1. purulent 4. productive 2. mucous 5. hemorrhagic 3. croupous 6. diphtheria 28. Microscopic characteristics of acute simple appendicitis 1. tricks of purulent inflammation in the mucosa 4. croupous inflammation 2. ulceration of the mucous membrane 5. plethora of blood vessels 3. catarrhal inflammation 29. Crohn's disease is 1. granulomatous disease of the small and large intestine 2. malignant tumor of the small intestine 3. fibrinous inflammation of the cecum 4. fibrinous inflammation of the small intestine 5. descending tuberculosis 30. Endophytic colorectal cancers often grow in 1. cecum 4. in the ascending region 2. transverse colon 5. rectum 3. sigmoid colon 31. When healing intestinal ulcers with typhoid fever in their place can be found 1. polyps 4. diverticulums 2. petrificates 5. star scars 3. small scars 6. foci of lymphoid tissue 32. Characterization of catarrhal colitis in dysentery 1. cerebral swelling of Peyer's plaques 2. diffuse leukocyte infiltration 3. hyperemia of the mucous membrane 4. mucosal necrosis 5. desquamation of the epithelium 33. With phlegmonous appendicitis, inflammation develops. 1. diphtheria 4. putrefactive 2. hemorrhagic 5. croupous 3. fibrinous 6. purulent 34. Clinical manifestations of Crohn's disease 1. bloody mucous diarrhea 4. abdominal pain 2. massive bleeding 5. fever 3. intestinal perforation 6. tenesmus 35. By the nature of the growth of exophytic cancers: 1. saucer-shaped 4. polypous 2. plaque-like 5. mushroom-shaped 3. ulcerative 36. With cholera, changes are mainly localized in the gut 1. skinny 4. sigmoid 2. blind 5. iliac 3. straight 6. transverse colonic 37. Complications of typhoid fever 1. pylephlebitic liver abscesses 2. typhoid sepsis 3. croupous penevmonia 4. focal pneumonia 5. bleeding 6. peritonitis 7. pleurisy 38. Microscopic characteristics of phlegmonous ulcerative appendicitis 1. vascular congestion 2. ulceration of the mucous membrane 3. leukocyte infiltration of all layers of the wall 4. leukocyte infiltration of only the mucous membrane 39. Macroscopic characterization of the intestine in Crohn's disease 1. thickening of the mesentery 2. narrowing of the intestinal lumen 3. dull serous membrane 4. expansion of the lumen of the intestine 5. circular ulcerative lesions 6. intermittent lesions 7. The mucosa looks like a “cobblestone pavement” 8. shiny and full-blooded serous membrane 9. ulcerative lesions oriented along the length of the intestine 40. By the nature of the growth of endophytic cancers 1. saucer-shaped 4. polypous 2. plaque-like 5. mushroom-shaped 3. ulcerative 6. diffuse - infiltrative 41. Characterization of the spleen in the algide period of cholera 1. lymphoid hyperplasia 2. atrophy of lymphoid tissue 3. white pulp amyloidosis 4. heart attacks 42. Common changes in typhoid fever 1. rash 4. focal pneumonia 2. hepatomegaly 5. spleen hyperplasia 3. encephalopathy 6. waxy necrosis of skeletal muscle 43. The reason for the primary gangrene of the appendix 1. necrosis of the muscle membrane 4. tricks of purulent inflammation of the mucosa 2. ulceration of the mucous membrane 5. leukocyte infiltrate in all layers 3. thrombosis of the appendicular artery of the appendix 44. Microscopic characteristics of the intestine in Crohn’s disease 1. giant cell granulomas 2. crypt abscessing 3. epithelial metaplasia 4. progressive mucosal atrophy 5. neutrophilic crypt infiltration 6. the presence of a fibrinous film 45. Precancerous colon diseases include 1. ulcerative colitis 2.
dysenteric ulcerative colitis 3. Crohn's disease 4. adenomatous polyps 5. intestinal strictures and stenoses 46. Morphological characteristics of the small intestine at the stage of cerebral swelling in typhoid fever 1. mucosal polyposis 4. purulent inflammation 2. mucosal necrosis 5. granulomatosis 3. sclerosis of the intestinal wall 47. The parts of the intestine that are most commonly affected by bacterial dysentery 1. appendix 5. 12 duodenal ulcer 2. jejunum 6. sigmoid colon 3. cecum 7. descending colon 4. rectum 8. ascending colon 48. Morphological characteristics of chronic appendicitis 1. atrophy of all layers 4. sclerosis of the process wall 2. ulceration of the mucosa 5. necrosis of the muscle membrane 3. catarrhal inflammation 49. Ulcerative colitis is 1. ulcerative infiltrative lesion of the colon 2. ulcerative proliferative lesion of the colon 3. necrotizing enterocolitis 4. granulomatous disease 5. colon tuberculosis 50. The most common variant of anorectal cancer 1. adenocarcinoma 4. squamous 2. mucous cancer 5. undifferentiated 3. cricoid - cell 51. Cause of severe diarrhea with cholera 1. extensive ulcerative defects 2. bowel lesions 3. the introduction of cholera vibrio 4. exposure to cholera enterotoxin 5. toxic damage to the nervous system of the intestine 52. Morphological changes in the intestine with typhoid fever in the first week of illness 1. the formation of clean ulcers 2. necrosis of Peyer's plaques 3. proliferation of granulation tissue 4. exudative inflammation in solitary follicles 5. hyperplasia of cells of a monocytic macrophage series in Peyer's plaques 53. Complications of acute appendicitis 1. process self-amputation 4. cystadenoma 2. wall perforation 5. mucocele 3. bleeding 54. Macroscopic characteristics of the colon with ulcerative colitis 1. hyperemia of the serous membrane 5. atrophy of the mucosa 2. mucosal hypertrophy 6. wall thickening 3. ulceration of the mucosa 7. pseudo-polyps 4. mucosal hyperemia 55. Hematogenous metastases of intestinal cancer are found in 1. lungs 4. adrenal glands 2. kidney 5. lymph nodes 3. liver 56. Dehydration in cholera is associated with 1. massive allocation of sodium and water from cells 2. high levels of intracellular AMP 3. fluid malabsorption 4. epithelial necrosis 57. Stages of local changes in typhoid fever 1. necrosis 5. cerebral swelling 2. healing 6. formation of clean ulcers 3. gastroenteritis 7. formation of dirty ulcers 4. fibrinous colitis 8. follicular ulcerative colitis 58. Pylephlebitic liver abscesses may occur with 1. duodenal ulcer 4. acute appendicitis 2. mesenteric artery thrombosis 5. dysentery 3. gastric ulcer 59. With ulcerative colitis, microscopic changes are presented 1. crypt abscessing 2. granulomas in the submucosal layer 3. “galloping” ulcerations of the mucosa 4. continuous ulcerations of the mucosa 5. inflammatory mononuclear infiltrate 60. Endophytic cancers often lead to 1. melene 4. anemia 2. to obstruction 5. bleeding 3. to bloating 61. At the autopsy of the deceased during the algide period, cholera can be detected 1. lack of rigor mortis 2. severe rigor mortis 3. cadaveric blood, liquid and light 4. cadaveric blood is thick and dark 5. extreme dehydration 62. Stages of dysenteric colitis 1. granulomatous 4. catarrhal 2. ulcer healing 5. serous 3. fibrinous 6. ulcerative 63. Purulent mesenteric vein thrombophlebitis may be complicated 1. lung abscesses 4. phlegmon appendix 2. liver abscesses 5. self-amputation of the appendix 3. phlegmon of the stomach 64. Intestinal complications of acute ulcerative colitis 1. pericholangitis 3. colorectal cancer 2. toxic dilatation 4. perforation of the intestinal wall 65. Exophytic cancers often lead to 1. melene 4. anemia 2. to obstruction 5. bleeding 3. to bloating 66. The stages of cholera 1. proctitis 4. algid period 2. sigmoiditis 5. serous enteritis 3. gastroenteritis 67. The reason for the development of ulcerative colitis in dysentery 1. intestinal gangrene 3. massive hemorrhage 2. necrosis of granulomas 4. rejection of the fibrinous film 68. During histological examination of the appendix against the background of diffuse wall infiltration, small intramural abscesses are determined. This is appendicitis. 1. phlegmonous ulcer 4. catarrhal 2. apostematous 5. putrefactive 3. chronic 69. Set Compliance Form of ulcerative colitis MORPHOLOGICAL SIGNS A) acute 1. fringed pseudo-polyps B) chronic 2. inflammatory polyps 3. mucosal atrophy 4. thickening of the intestinal wall 5. thinning of the intestinal wall 6. fields of fibrosis 70. Morphological variants of intestinal adenomas 1. mucous membranes 4. villous 2. fibrous 5. small cell 3. tubular 71. Specific complications of cholera 1. splenomegaly 4. cholera meningitis 2. ulcerative gastritis 5. post-cholera uremia 3. cholera typhoid 72. The preferred route of transmission of bacterial dysentery 1. airborne 4. parenteral 2. fecal-oral 5. pin 3. transmission 73. Pilephlebitic liver abscesses in acute appendicitis are a consequence of 1. thromboembolism from the appendicular artery 2. self-amputation of the appendix 3. inflammation of the veins of the appendix 4. perforation of the appendix 74. Intestinal complications of Crohn's disease 1. fistulas between the intestines 4. fibrous strictures 2. intestinal obstruction 5. ulcerative defects 3. protein-free enteropathy 75. The path of infection with typhoid fever 1. airborne 4. parenteral 2. fecal-oral 5. pin 3. transmission ANSWERS No. | the answers | No. | the answers | No. | the answers | | 3 | 26. | 4 | 51. | 4 | | 1, 2, 5 | 27. | 6 | 52. | 5 | | 4 | 28. | 3, 5 | 53. | 12 | | 1, 3, 5, 6 | 29. | 1 | 54. | 3, 4, 5, 7 | | thirteen | thirty. | 3, 5 | 55. | thirteen | | 1, 2, 3 | 31. | 3, 6 | 56. | 1, 2, 3 | | 2, 4, 6 | 32. | 2, 3, 5 | 57. | 1, 2, 5, 6, 7 | | 3, 4, 6 | 33. | 6 | 58. | 4, 5 | | 2 | 34. | 4, 5 | 59. | 4, 5 | | 1, 2, 5 | 35. | 2, 4, 5 | 60. | 2, 3 | | 1, 2, 6 | 36. | fifteen | 61. | 2, 4, 5 | | 2, 3, 4, 5 | 37. | 2, 4, 5, 6 | 62. | 2, 3, 4, 6 | | 2, 4, 5 | 38. | 1, 2, 3 | 63. | 2 | | 2, 3 | 39. | 1, 2, 3, 6, 7, 9 | 64. | 2, 4 | | 1, 2, 3, 4 | 40. | 3, 6 | 65. | 1, 4, 5 | | 3, 5 | 41. | 2 | 66. | 3, 4, 5 | | 2 | 42. | 1, 5, 6 | 67. | 4 | | 3 | 43. | 3 | 68. | 2 | | 1, 3, 4 | 44. | 2, 3, 4, 5 | 69. | A-1,2,5; B-2,3,4,6 | | 2 | 45. | 1, 3, 4 | 70. | 3, 4 | | 3, 6 | 46. | 5 | 71. | 3, 5 | | 2, 3, 5 | 47. | 4, 6, 7 | 72. | 2 | | 2, 4 | 48. | 14 | 73. | 3 | | 12 | 49. | 1 | 74. | 1, 3, 4 | | 1, 2, 4 | fifty. | 4 | 75. | 2 |
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| Bowel disease. Infectious enterocolitis (dysentery, typhoid fever, cholera). Nonspecific ulcerative colitis. Crohn's disease. Coronary bowel disease. Appendicitis. Colon cancer
| - Inflammatory bowel disease (ulcerative colitis, Crohn's disease)
Inflammatory bowel disease (ulcerative colitis, disease
- Coronary bowel disease.
Coronary bowel disease (IBS) is a disease manifested by various forms of intestinal ischemia, in which the affected segment of the intestine receives less blood than is required to maintain its structure and function. The main causative factors are vascular occlusion and hypotension, which together can lead to dysregulation of mesenteric blood flow. The reasons can be divided into
- Large intestine inflammation (colitis)
Colitis is a disease resulting from inflammatory-dystrophic changes in the colon. The inflammatory process can be limited or spread over several segments. The most commonly affected area is the distal colon (proctitis) or the rectum and sigmoid colon (proctosigmonditis). There are acute and chronic colitis. The most common colitis is non-infectious,
- Intestinal infections (typhoid fever, paratyphoid A and B, dysentery, cholera) and their prevention.
Acute intestinal infections include dysentery, typhoid fever, paratyphoid A and B, cholera infectious hepatitis, etc. These diseases are characterized by the same location (intestines), the same transmission route (fecal-oral), similar symptoms (disorders of the intestinal tract). Diseases are caused by pathogenic bacteria that are transmitted from a sick person to a healthy person.
- The role of the large intestine in the life of vegetarians and meat-eaters (Mechnikov, Shatalova and large intestine)
"For a long time, scientists could not understand what role the large intestine plays in our body. I. Mechnikov even thought that we simply did not need such an organ." “There is nothing surprising in the fact that Ilya Ilyich Mechnikov underestimated the role of the colon. Indeed, if the body receives essential amino acids with animal proteins, and nobody functions of the large intestine
- DIGESTIVE DISEASES. INTESTINAL DISEASES
Bowel diseases are extremely diverse, they relate to selective lesions of the small or large intestines, but are often accompanied by a combined lesion of all parts of the intestine. In the structure of diseases and mortality from them, cardiovascular invariably occupy the first place, and among them - atherosclerosis and hypertension. It has long been identified as an independent nosological form
- III. INTESTINAL CANCER
Colon cancer currently occupies 3rd place among its other locations and, according to various authors, makes up 98-99% of all intestinal cancers. Colon cancer is twice as likely to affect men than women. The most common tumor location is the sigmoid (25-30%) and, especially, the rectum (about 40%). All other parts of the colon are significantly affected by the cancer.
- The structure and functions of the small and large intestine. Congenital malformations of the intestine
In an adult, the length of the small intestine reaches about 6 m, and the large - 1.5 m. In addition to the width and structure of the outer shells, the small intestine differs from the thick structure of the mucous membrane, in which circular folds, intestinal villi and crypts (intestinal glands). The villi protrude into the lumen in the form of finger-shaped outgrowths covered with a cylindrical epithelium. Similar to
- Peptic ulcer of the stomach and duodenum
Questions for repetition: 1. Methods of examination of children with diseases of the stomach and 12 duodenal ulcer. 2. Fractional study of gastric secretion in children. Test questions: 1. Modern views on the etiopathogenesis of peptic ulcer. 2. Classification of peptic ulcer. 3. Clinical manifestations of duodenal ulcer: 3.1. pain syndrome 3.2. dyspeptic syndrome 3.3.
- Bowel disease
The main clinical symptoms: diarrhea, anarexia, pain, flatulence, lack of bowel movement, polydipsia, dehydration. Change in the shape, color, quantity and volume of feces. Sometimes vomiting. Diarrhea is the main symptom of bowel disease. Even a once unformed stool is considered a diarrhea. Constipation is the absence of bowel movements within 2 days. The strong smell of feces does not indicate severity
- DISEASES OF THE STOMACH AND GUT
Diseases of the gastrointestinal tract are very diverse, numerous and include diseases of the pharynx, salivary glands, esophagus, stomach, intestines, pancreas, liver and gall bladder. However, peptic ulcer, stomach cancer, appendicitis and liver diseases are of the greatest importance in human pathology. ULCER DISEASE is a common chronic, cyclically occurring disease with frequent
- GASTRIC AND TWELVE ULCER DISEASES
Since about 200 years ago, Crewellier attracted the attention of doctors to gastric ulcers, interest in this disease has been steadily increasing. Approximately the same applies to duodenal ulcer described in detail much later (Moynihan, 1913). Peptic ulcer is now understood as a common, chronic, recurring, cyclically occurring disease in which
- Early active detection of bowel disease
Questionnaires include signs of dyspepsia, diarrhea, constipation, and blood excrement with feces. Screening of the 1st level includes analysis of feces for digestibility, occult blood, digital examination of the rectum. The further examination program is determined after a physical examination. Endogenous risk factors for colon cancer: the nature of nutrition (a combination in the diet of coarse fiber with
- Question 21: ULCER OF THE STOMACH AND TWELVE
—Chronic recurrent disease in which, as a result of disturbances in the nervous and humoral mechanisms that regulate secretory-trophic processes in the gastroduodenal zone, an ulcer forms in the stomach or duodenum (less often two or more ulcers). Etiology, pathogenesis. Peptic ulcer is associated with a violation of the nervous, and then the humoral mechanisms that regulate the secretory,
- Peptic ulcer of the stomach and duodenum (ciphers K 25; K 26)
Definition Peptic ulcer of the stomach and (or) duodenal ulcer (essential mediogastric and duodenal ulcer) - a heterogeneous disease with a lot? actor etiology, complex pathogenesis, chronic relapsing course, morphological equivalent in the form of a mucosal and submucosal defect with an outcome in the connective tissue scar. Statistics. In different age periods
- Bowel disease
Appendicitis Appendicitis of the cecum. воспаление червеобразного отростка • Различают две клинико-морфологические формы аппендицита: острый и хронический. Острый аппендицит * В развитии имеют значение: а. Обструкция аппендикса (чаще каловыми массами) со снижением резистентности слизистой оболочки и инвазией микроорганизмов в стенку отростка. b. Необструктивный аппендицит может
- The composition of the microflora of the large intestine. The functions of the microflora of the large intestine
A detailed composition of the intestinal microflora is given in Appendix 1. The entire intestinal microflora is divided into: - obligate (main microflora); - optional part (conditionally pathogenic and saprophytic microflora); Obligatory microflora. Bifidobacteria are the most significant representatives of obligate bacteria in the intestines of children and adults. These are anaerobes, they do not form a dispute and
- Болезни желудка и кишечника
1. Определение гастрита: а) дистрофическое заболевание слизистой оболочки желудка б) воспалительное заболевание слизистой оболочки в) дисрегенераторное заболевание слизистой желудка г) инфекционное заболевание с поражением слизистой оболочки желудка д) предраковое заболевание желудка Правильный ответ: б 2. Сущность морфологических изменений при остром гастрите: а) экссудативное воспаление
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