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Pathogenetic and symptomatic therapy.



To stop the allergic component of the disease, antihistamines, non-specific anti-inflammatory drugs are used. Antihistamines are also prescribed to all patients for the period of etiotropic therapy with antiparasitic drugs, since the breakdown of helminths enhances the allergic reactions of the patient's body.

With severe intoxication, infusion detoxification therapy with glucose-salt solutions is carried out. In the absence of pronounced intoxication phenomena during the period of etiotropic therapy, oral detoxification is recommended.

With severe hypoproteinemia, parenteral administration of a solution of albumin and plasma is indicated.

In severe forms of trichinosis, neurocysticercosis with signs of arachnoiditis, vasculitis, glucocorticosteroids (GCS) are used. Carrying out glucocorticosteroid therapy leads to disruption of the encapsulation process of trichinella larvae in the muscles, which leads to a protracted and recurrent course of trichinosis. An absolute indication for the appointment of corticosteroids is an extremely severe course of invasion with the development of life-threatening syndromes (toxic toxic shock, encephalopathy, etc.), as well as the occurrence of myocarditis. Prednisone with myocarditis is prescribed in a dose of 20-60 mg per day until the main clinical and laboratory ones (cardiac deafness, tachycardia, arrhythmia, cardiac enlargement, hyperfermentemia - CPK, LDH1-5, AsAT) and electrocardiographic signs of complication are stopped.

In the acute period of cysticercic encephalitis, anticonvulsant and symptomatic drugs are prescribed at the beginning of therapy, and then anthelmintics in combination with glucocorticosteroids.

Surgical treatment is carried out with echinococcosis, cysticercosis (in cases of hydrocephalus, cysticercosis of the ventricles of the brain and damage to the spinal cord), in severe (noncurable) cases of ocular toxocariasis.

Before carrying out surgical treatment, patients with echinococcosis are recommended to conduct one course of etiotropic therapy.
The volume of surgical intervention is determined by the prevalence of the pathological process and the presence of complications. A single cyst can be removed or drained under the control of ultrasound with the introduction of 95% ethanol with mebendazole into the cavity (PAIR method: puncture-aspiration-instillation-re-aspiration). “Ideal echinococcectomy” involves the extraction of a cyst without compromising the integrity of the cuticular membrane and fibrous capsule. With a common process, a part of the organ is resected. In cases of alveococcosis, surgical intervention is especially necessary for damage to the lungs and bones. The use of nemozole increases the survival of patients after radical surgery, its purpose is also justified in inoperable cases.

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Pathogenetic and symptomatic therapy.

  1. Symptomatic therapy
    In the complex treatment of influenza and acute respiratory infections in children, the following drugs are used for symptomatic therapy (see the Appendix to Chapter 6, tables 6.11–6.17). Antipyretic drugs. It must be remembered that the danger to children of a number of antipyretic drugs has been proven. In children, amidopyrine, antipyrine, phenacetin, aspirin, analgin, salicylamide are not used, but as antipyretic
  2. Symptomatic therapy
    The disease includes many different symptoms and complications that reduce the quality of life of patients. The most common symptoms in MS are: impaired sensitivity, dizziness, pathological chronic fatigue, dysfunction of the pelvic organs, spasticity. Therapy to relieve or alleviate the symptoms of the disease can significantly improve the quality of life of patients.
  3. PATHOGENETIC THERAPY OF MULTIPLE SCLEROSIS
    PATHOGENETIC THERAPY OF SCATTERED
  4. Symptomatic treatment
    Symptomatic treatment is understood to mean all treatment methods that contribute to the removal or mitigation of the symptoms of the disease and subsequent conditions caused by the disease, but do not eliminate its causes. In multiple sclerosis, symptomatic treatment not only directly mitigates the manifestation of sufficiently unpleasant symptoms, but also indirectly contributes to the implementation of rehabilitation measures, which in the first place
  5. Symptomatic Anemia
    The development of anemia is possible with a number of pathological conditions that would seem to be not related to the hematopoietic system. Diagnostic difficulties, as a rule, do not arise if the underlying disease is known and the anemic syndrome does not prevail in the clinical picture. The significance of symptomatic (secondary) anemia is explained by their relative frequency in pediatrics and possible resistance to therapy.
  6. SYMPTOMATIC IRON DEFICIENCY ANEMIA, CHLORANEMIA
    Symptomatic iron deficiency anemia develops against the background of a certain etiological factor: chronic enteritis, chronic nephritis (chloranemia of the "brother"), due to resection of the stomach (agastric chloranemia), occult blood loss (with hiatal hernia, "herniated, anemia) cancerous hyposiderosis), a chronic infection
  7. Symptomatic arterial hypertension
    SYMPTOMATIC, OR SECONDARY, ARTERIAL HYPERTENSION (hypertension) is hypertension, causally associated with certain diseases or injuries of the organs (or systems) involved in the regulation of blood pressure. It is detected in 5-15% of patients suffering from hypertension. Classification. There are many classifications of SG, however, there are four main groups of SG. I. Renal (nephrogenic). II. Endocrine.
  8. Symptomatic hypertension
    570. DOES NOT RESULT IN THE DEVELOPMENT OF VASORENAL ARTERIAL HYPERTENSION 1) Takayasu’s disease 2) atherosclerosis of the renal arteries 3) Alport syndrome 4) fibromuscular dysplasia of the renal arteries 119 5) tumors leading to extravasal compression of the renal artery 571. ANIMAL ) renal vein catheterization with
  9. Clinical and pathogenetic forms of AF (TP)
    From a clinical point of view, the following forms of AF (TP) (classification) can be distinguished: - Chronic is not yet completely clear, some results of surgical effects on the corresponding atrial zones aimed at eliminating TP are encouraging. The last point of this classification seems to us especially important, since CVS and WPW syndrome significantly alter the clinical course and
  10. SYMPTOMATIC AG OF ENDOCRINE ORIGIN
    SYMPTOMATIC AG OF ENDOCRINE
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