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Training target: using diagnostic algorithms, be able to establish a diagnosis of mumps, determine the clinical form of the disease, complications and prescribe adequate treatment.

Assignment for independent study of the topic.

Using a textbook and lecture material to acquire the necessary basic knowledge, learn the following sections for practical training:

1. Etiology, pathogenesis, clinical manifestations of mumps and its complications.

2. Clinical and laboratory diagnosis of mumps and lesions of the salivary glands of another etiology.

3. Criteria for the differential diagnosis of mumps and lesions of the salivary glands of another etiology.

4. Principles and methods of treating patients with uncomplicated and complicated forms of mumps.

Test your knowledge by answering the following questions:

- pathophysiological mechanisms of the occurrence of organ damage in mumps;

- syndromes and symptoms characterizing mumps;

- clinical and laboratory data on which the diagnosis of mumps is based;

- The most common complications of mumps;

- clinical and laboratory data showing the development of complications in a patient with mumps;

- ways to prevent the development of possible complications of mumps;

- The main methods and means of treating patients with uncomplicated and complicated forms of mumps.

The solution of situational clinical problems N ......

Materials for independent preparation for a practical lesson on the stages of diagnosis and treatment. In order to clarify the indicative basis of actions during the examination of patients with mumps, analyze the algorithms, schemes and tables located in accordance with a phased diagnostic treatment search.

The algorithm for the diagnosis of mumps.

Questions for self-control:

- The symptoms characterizing the syndrome of general infectious intoxication in patients with mumps;

- features of the temperature curve in uncomplicated and complicated forms of mumps;

- salivary glands affected by the causative agent of mumps;

- clinical differences in salivary gland inflammation in mumps of purulent mumps, from lymphadenitis;

- additional laboratory and allergological methods used to diagnose mumps.

The algorithm for determining the clinical form of mumps

Questions for self-control:

- criteria for assessing the severity of the condition (clinical form) with mumps;

- the period of the disease in which complications arise;

- the possibility of complications with a mild form of mumps;

- complications and their role in changing the degree of suitability for military service, access to flights of flight-lifting personnel, scuba diving and diving descents, who have had complicated forms of mumps.

Differential diagnosis of lesions of the parotid salivary glands

Note: in cases of edema of the subcutaneous tissue of the neck in the area of ​​the parotid salivary glands or submandibular salivary glands, mumps must be differentiated from toxic pharyngeal diphtheria; with toxic diphtheria, the syndrome of general infectious intoxication is more pronounced, the retromandibular fossa is free from edema, regional lymphadenitis is noted, and with pharyngoscopy, swelling of the pharyngeal mucosa and fibrinous deposits with spread beyond the palatine tonsils are determined.

An algorithm for determining the complications of mumps

Note: with mumps, in addition to inflammation of the testicles and pancreas, lesions of other glands of ectodermal origin (mammary, thyroid, prostate, Bartholin's, ovaries) may develop; rare complications of mumps are labyrinthitis, auditory nerve neuritis, arthritis.

Questions for self-control:

- differential diagnostic signs that distinguish between serous meningitis caused by the mumps virus and the causative agent of tuberculosis;

- studies to help identify pancreatic inflammation in mumps;

- the nature of the temperature curve in complicated forms of mumps;

- the pathogenesis of complications in mumps.

The treatment regimen for patients with mumps

Questions for self-control:

- indications for the appointment of patients with mumps gamma globulin, antiviral drugs;

- therapeutic tactics of a doctor in acute pancreatitis;

- residual effects of mumps;

- rules for extracting convalescents after mumps; military-medical expert decision of the military-medical commission (medical-flight commission) in relation to patients with mumps (uncomplicated and complicated course).

Tasks for self-monitoring in the clinical evaluation of laboratory tests

Evaluate a general clinical study of the cerebrospinal fluid of a patient with mumps complicated by serous meningitis:

color and transparency colorless, transparent

pressure 300 mm aq

cytosis 450 cells in 1 μl

neutrophil cytogram - 46%

lymphocytes - 54%

protein 0.99 g / l

Pandy reaction +

Nonne-Apelt reaction +

sugar 2.9 mmol / l (in blood - 5.6 mmol / l)

chlorides 117 mmol / l

no fibrin film

microflora of sediment -

Evaluate the activity of amylase in the blood and urine of a patient with mumps: in the blood (Caraway method) - 150 mg / (s l); in the urine - 630 mg / h ml.

Prescribe recipes: furatsillina solution for rinsing the mouth, contracal for intravenous administration, analginum inside.
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  1. Parotitis
    Mumps (mumps) - refers to acute contagious viral diseases, characterized by fever, general intoxication, an increase and soreness of the salivary glands, mainly the parotid. Etiology. The virus causative agent of mumps infection is unstable, is inactivated by heating (at a temperature of 70 ° C for 10 min), ultraviolet radiation, exposure to formalin solutions
    C. George Ray (C. George Ray) Definition. Mumps (mumps) is an acute highly contagious disease of viral etiology, characterized by an increase in the parotid salivary glands, and sometimes involvement in the process of the sex glands. meninges, pancreas, and other organs. Etiology. The causative agent of mumps belongs to the family
  3. Parotitis
    Mumps (mumps) is a childhood viral infection characterized by acute inflammation in the salivary glands. The causative agent of mumps belongs to the RNA-containing viruses, the genus Paramyxovirus. Infection occurs by airborne droplets. The susceptibility to this disease is about 50-60% (that is, 50-60% of those who were in contact and did not get sick or not vaccinated get sick). Piggy
  4. Parotitis.
    Mumps (mumps) is an acute infectious disease with a primary lesion of the salivary glands. The causative agent is the Pneumophilis parotidis RNA virus from the genus paramyxoviruses. Infection occurs from a patient with manifest or, more often, erased and asymptomatic forms of the disease. The route of infection is airborne. Transmission of the virus through toys and household items infected
  6. Question 28 The epidemic process
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  7. Epidemic Process Basics
    The epidemic process is the occurrence and spread of infections among the population. For the emergence and continuous course of the epidemic process, the interaction of three factors is necessary: ​​the source of the causative agents of the infection, the transmission mechanism of the infection, and the susceptible population. Shutting down any of these links interrupts the epidemic process. The biological basis of the epidemic
  8. Epidemic lethargic encephalitis Economo
    Economo's epidemic lethargic encephalitis (synonym: epidemic encephalitis type A, "sleeping" disease) was first recorded in 1915 by the troops near Verdun and described in 1917 by the Austrian neuropathologist K. Economo. The disease in those years proceeded in the form of epidemics that swept many countries of the world. In subsequent years, the disease manifested itself sporadically. Current disease in a typical
    Peter L. Ferine Definition. Epidemic relapsing fever is an acute infection characterized by repeated cycles of a rise in body temperature, which are separated from each other by asymptomatic intervals of apparent recovery. It is caused by spirochetes of the genus Borrelia and is represented by two epidemiological varieties - a disease transmitted by lice and a disease transmitted
  10. Piglet Epidemic Diarrhea
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  11. Epidemic encephalitis
    (Economo's disease, sleeping sickness, lethargic encephalitis, primary non-purulent inflammation of the brain). The causative agent of this disease is a filtered virus, which has not yet been isolated. Epidemic encephalitis was first described by Economo in 1917 during an epidemic in Austria, in the USSR — A. I. Geymanovich (Kharkov) and Y. M. Raimistom (Odessa) during an outbreak in 1 & 19
  12. Cerebrospinal epidemic meningitis
    Etiology and pathogenesis The disease is caused by meningococcus Frankel-Vekselbaum. This is a gram-negative diplococcus, located most often intracellularly. 4 strains of the pathogen were isolated: A, B, C, D. The disease is transmitted by airborne droplets and contact through objects that were in the patient’s use. The entrance gate is the mucous membrane of the pharynx and nasopharynx. Get sick
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