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Erysipeloid (swine erysipelas, creeping erythema) is an acute slowly developing infectious skin lesion related to occupational diseases and usually resulting from a penetrating wound during manual processing of animal products. Purple-red, clearly defined rashes localized on the fingers and hands are noted. May be complicated by arthritis. In rare cases, severe endocarditis develops with damage to the aortic valve.
The main pathogens
Erysipelotrix (E.rhusiopathiae) is a ubiquitous gram-positive saprophytic bacillus.
Drugs of choice: benzylpenicillin. With endocarditis in large doses.
Alternative drugs: imipenem.
It should be remembered that E.rhusiopathiae is resistant to vancomycin.
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- LYSTERIOSIS AND ERISIPELOID
Paul D. Hoeprich (Paul D. Hoeprich) Listeriosis Definition. Listeriosis is caused by listeria (Listeria monocytogenes) and is manifested by many clinical syndromes. In most cases, this is a perinatal infection, in which the child becomes infected either through the placenta or during childbirth, passing through the birth canal. Etiology. Listeria is a gram-positive, non-acid resistant
- PIG HORSE
Erysipelas (Latin - Erysipelas suum, Erysipelothrix rhusiopathiae, Rhusiopathia suia; English - Swine Erysipelas, Diamond disease; erisipeloid) is an acute disease of young pigs characterized by fever, septicemia and inflammatory erythema of the skin, and in chronic and arthritis, endocarditis see color insert). Historical background, distribution, hazard and damage.
Cellulite is a diffuse acute inflammation of the skin and / or subcutaneous tissue, characterized by their compaction, hyperemia, leukocyte infiltration and edema without cell necrosis or suppuration, often accompanied by pain and fever, lymphangiitis and regional lymphadenopathy. A variety of pathological processes (impetigo, erysipeloid,
- Vizer V.A .. Lectures on therapy, 2011
On the subject - almost completely cover the difficulties in the course of hospital therapy, the issues of diagnosis, treatment, as stated, are concise and quite affordable. Allergic lung diseases Joint diseases Reiter’s disease Sjogren’s disease Bronchial asthma Bronchiectasis Hypertension Glomerulonephrosafasdit Esophageal hernia Destructive lung diseases
- ALLERGIC LUNG DISEASES
In recent decades, a significant increase in the number. patients with allergic diseases of the bronchopulmonary apparatus. Allergic lung diseases include exogenous allergic alveolitis, pulmonary eosinophilia, and drug
- EXOGENOUS ALLERGIC ALVEOLITES
Exogenous allergic alveolitis (synonym: hypersensitive pneumonitis, interstitial granulomatous alveolitis) is a group of diseases caused by intensive and, more rarely, prolonged inhalation of antigens of organic and inorganic dusts and are characterized by diffuse, in contrast to pulmonary eosinophilia, lesions of the alveolar and interstitial structures of the lungs. The emergence of this group
1. General measures aimed at disconnecting the patient from the source of antigen: compliance with sanitary and hygienic requirements at the workplace, technological improvement of industrial and agricultural production, rational employment of patients. 2. Drug treatment. In the acute stage, prednisone 1 mg / kg per day for 1-3 days, followed by a dose reduction in
- Chronic Eosinophilic Pneumonia
It differs from Leffler's syndrome in a longer (more than 4 weeks) and severe course up to severe intoxication, fever, weight loss, the appearance of pleural effusion with a high content of eosinophils (Lehr-Kindberg syndrome). The prolonged course of pulmonary eosinophilia, as a rule, is the result of under-examination of the patient in order to identify its cause. Besides the reasons
- PULMONARY EOSINOPHILIA WITH ASTHMATIC SYNDROME
This group of diseases can include bronchial asthma and diseases with a leading broncho-asthmatic syndrome, which are based on other etiological factors. These diseases include: 1. Allergic bronchopulmonary aspergillosis. 2. Tropical pulmonary eosinophilia. 3. Pulmonary eosinophilia with systemic manifestations. 4. Hyperoseosinophilic
1. Respiratory diseases: A guide for doctors: In 4 volumes. Edited by N.R.Paleeva. T.4. - M .: Medicine. - 1990. - S. 22-39. 2. Silverstov V.P., Bakulin M.P. Allergic lung lesions // Klin.med. - 1987. - No. 12. - S.117-122. 3. Exogenous allergic alveolitis / Ed. A.G. Khomenko, St. Mueller, V. Schilling. - M.: Medicine, 1987. -
- BRONCHEECTATIC DISEASE
Bronchiectatic disease is an acquired (in some cases congenital) disease characterized by a chronic suppurative process in irreversibly changed (enlarged, deformed) and functionally defective bronchi, mainly in the lower parts of the lungs. ETHIOLOGY AND PATHOGENESIS. Bronchiectasis are congenital in 6% of cases, as a fetal malformation, a consequence
- CLASSIFICATION OF BRONCHEKTASAS
(A.I. Borokhov, N.R. Paleev, 1990) 1. By origin: 1.1. Primary (congenital cysts) bronchiectasis. 1.1.1. Single (solitary). 1.1.2. Multiple. l..l-Z. Cystic lung. 1.2. Secondary (acquired) bronchiectasis. 2. In the form of bronchial enlargement: 2.1. Cylindrical. 2.2. Saccular. 2.3. Spindle-shaped. 2.4. Mixed. 3. The severity of the course
1. Respiratory diseases. Manual for Doctors Edited. N.R.Paleeva. - M .: Medicine, 1990. - T.Z., T. 4. 2. Okorokov A.N. Treatment of diseases of internal organs: Practical guidance: In З. TI - Mn. Vysh.shk., Belmedkniga, 1997. 3. Harrison T.R. Internal illnesses. - M .: Medicine, T.7,
- DISEASE (SYNDROME) OF THE RATER
Reiter’s disease (Reiter’s syndrome, Fissenge-Leroy’s syndrome, urethro-oculo-synovial syndrome) is an inflammatory process that develops in most cases in close chronological connection with infections of the genitourinary tract or intestines and manifested by the classical triad - urethritis, conjunctivitis, arthritis. Most often, young (20 - 40) men who have undergone urethritis are ill. Women, children and the elderly
- SHEHREN'S DISEASE (SYNDROME)
The combination of dry keratoconjunctivitis, xerostomia, and chronic polyarthritis was described in such detail by the Swedish ophthalmologist Shegren (Shegren, 1933) that soon attracted the attention of clinicians from different countries to this very peculiar clinical phenomenon, although isolated observations of such a triad or individual manifestations of secretory glandular insufficiency were previously described. Behind