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Tetanus

ETIOL. Clostridium tetani. AF: exotoxin (tetanospazmin - neurotoxin and tetanohemolysin). IST: soil, feces herbivores. is alive. and people. Thief: wounds, burns, frostbite, childbirth, abortion.

PZ required conv. - lack of O2. Input gate - 1) tetanospazmin - dvig. fiber perip. nerves + hematogenous - NS - paralysis standing up. neur. polysynaptic refl arc - violated. percent braking - convulsions. 2) tetanohemolysin - hemolytic. and cardiotoxic. d.

SP. 7-20days

KL. Forms: 1) generalizations; 2) local (lyceums, or goals); 3) Brunner's tetanus; 4) neonatal. Consciousness is always clear. Started per: pain in the region of the wound, fibril twitching of muscles near, contraction of the muscles with palpation above the wound. Generalized .: acute, 3 leading symptoms: 1) Trism is chewed. muscle 2) a sardonic smile; 3) dysphagia. Hypertonicity of muscles from top to bottom, opisthotonus; pain, cramps, disturbance. resp., tachycardia, sweating,? t up to 42o. Local: rarely, damage to the muscles of the wound, then their hypertonicity and tetanic. cramps, then gradual generalization. process. 1) the front tetanus of Rosa with head injuries; 2) local spasm and cramps throat. the muscles. S. Brunner: defeated. top departments of the spin. and continued. brain, then generalization. Neonatal: inconsistency of symptoms. Always obvious hypertonicity and cramps.

Severity. 1) mild (IP-20 days, symptom in 5-6 days, hypertonicity died, insignificant cramps, t - normal / subfebri; 2) medium (IP-15-20 days, symptom in 3- 4 days, hypertonicity died., Spasms are infrequent, died., T - high, tachycardia; 3) severe (IP-7-14 days, symptom.
for 1-2 days, express hypertonic intensity cramps, t - high, tachycardia, sweating); 4) very severe (PI? 7 days, symptoms of lightning fast, t-42o, severe hypertonicity, frequent cramps, which means tachycardia, tachypnea, cyanosis).

DIAG. Bacteriological bacterioscopic., biological. (test on mice).

D / DIAG. Trism: will inflame processes in the lower. jaw and its joints, peritoneal gland., with peritonsillitis. Convulsions: hysteria, epilepsy, strychnine poisoning. Tetanus Rose diff. from stem encephalitis with, cat. no trism.

LECH. In the special. centers. 1) surgeon. processing wounds with the introduction of 3000-10000 IU p / column into their area. Serving .; 2) antitoxic. p / post. syv. 100,000 - 150,000 IU (adult), 20,000 - 40,000 IU (new), 80,000 - 100,000 IU (children) once; 3) p / post. immunoglobulin 900 IU once; 3) tetanus toxoid 1 ml / m every 3-5 days; 4) an anti-vessel. - chlorpromazine (3 mg / kg), sibazon (1-3 mg / kg), lytic mixture (chlorpromase 2.5% 1 ml, diphenhydramine 1% 2 ml, promed 2% 1 ml, scopolamine 0.05% 1 ml), or neuroleptanalgesis (with heavy.f.); 5) A / B - warning. complicated Infusion. therapy.

Donkey Pneumonia, sepsis, compress. fracture tel., breaks and contract. muscle paralysis III, IV, VII.

EXTRA PROF. Priv: post. toxoid 0.5 ml. Nepriv: 1 ml post. toxoid + 3000ME p / column. syv. (other syringe to another part of the body
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Tetanus

  1. Tetanus
    Tetanus is an acute infectious disease caused by the action of the toxin secreted by the bacillus of Nicolaera (Clostridium tetani), a microorganism that can remain viable for many years while in the ground. This bacillus easily penetrates the human body through a wound, especially if dirt enters the wound. See the article SHOOTS, taking into account that the metaphysical meaning of tetanus
  2. Tetanus
    Tetanus is an acute toxemia caused by the action of exotoxin (tetanospasmin) produced by the bacteria Clostridium tetani. The toxin is produced by the vegetative forms of the microorganism at the place of its penetration into the body tissue, and then enters the central nervous system and is fixed there. Etiology. Tetanus causative agent - obligate anaerobic, thin gram-positive motile
  3. TETANUS
    Tetanus [Greek - Tetanus (curing); English - Lockjaw] - an acute, non-contagious wound toxicoinfectious disease of mammals of animals, birds and humans, characterized by increased reflex excitability, convulsive tonic convulsions of the muscles of the body under the influence of the toxin of the pathogen (see color insert). Historical background, distribution, hazard and damage.
  4. Tetanus
    Tetanus is an acute infectious disease characterized by tonic tension of skeletal muscles and periodic generalized seizures, which is associated with damage to the motor structures of the central nervous system by the toxin of the pathogen. The causative agent of tetanus has the ability to produce the strongest exotoxin, which is associated with clinical manifestations
  5. TETANUS (TETANUS)
    Tetanus is an acute wound infectious disease of animals and humans, characterized by damage to the nervous system, reflex excitability and convulsive contraction of the muscles of the body without disturbing consciousness. Etiology. The causative agent of tetanus is widespread in nature, there are many in the soil of gardens and vegetable gardens, in manure. There is evidence that it multiplies in the intestines of animals, with their feces
  6. Tetanus
    ETIOL. Clostridium tetani. AF: exotoxin (tetanospazmin - neurotoxin and tetanohemolysin). IST: soil, feces herbivores. is alive. and people. Thief: wounds, burns, frostbite, childbirth, abortion. PZ required conv. - lack of O2. Input gate - 1) tetanospazmin - dvig. fiber perip. nerves + hematogenous - NS - paralysis standing up. neur. polysynaptic refl arc - violated. percent braking - convulsions. 2)
  7. Tetanus
    Scope of examination 1. Tetanus is an acute infectious disease characterized by toxic damage to the nervous system, manifested by tonic convulsions. The causative agent of the disease is the anaerobic spore-forming bacillus of Clostridium tetani. 2. Entrance gates are wounds or other damage to the skin and mucous membranes, including burns, frostbite, scuffs, criminal abortions,
  8. Tetanus
    Tetanus is an infectious disease caused by the neurotropic toxin of anaerobic tetanus bacillus, characterized by attacks of tonic seizures. In newborns, the infection penetrates through the umbilical wound, in older children through any wound, especially a contaminated one. Extremely dangerous contaminated ground stabbed and lacerated wounds with crushed tissues, due to the danger of anaerobic
  9. TETANUS
    Tetanus is an acute wound toxicosis of animals caused by Clostridium tetanus and characterized by damage to the nervous system, reflex excitability and convulsive muscle contraction without impaired consciousness. The causative agent lives mainly in soil containing manure. There is evidence that pathogenic clostridia, multiplying in the intestines of animals, are excreted into the external environment along with feces.
  10. TETANUS
    Harry H. Beaty Definition. Tetanus is an acute, often fatal, disease caused by endotoxin produced by bacteria entering the wound (Clostridium tetani). Generalized increase in rigidity and convulsive spasms of skeletal muscles are typical for it. Etiology. Tetanus bacillus is strictly anaerobic gram-positive motile
  11. TETANUS
    An acute wound infectious disease characterized by increased reflex excitability, tonic convulsive contractions of all or certain muscle groups of the body under the influence of CI toxin. tetani, formed at the site of the pathogen penetration into the body. All types of mammals are susceptible to tetanus, mostly horses, then sheep, goats, cattle and
  12. Tetanus
    Tetanus
  13. tetanus (pathogenesis, clinical features, diagnosis, treatment)
    Tetanus (tetanus) is an acute infectious disease caused by exposure to tetanus bacillus exotoxin with a predominant lesion of the nervous system, characterized by tonic and convulsive contractions of striated muscles. Etiology: Clostridium tetani - in the external environment exists in the form of extremely stable spores, which, under favorable anaerobic
  14. Infections of the external integument
    Tetanus is an acute disease with symptoms of intoxication of the central nervous system, tonic and clonic muscle cramps. Epidemiology. Pathogen - tetanus bacillus, strict anaerobic; the vegetative form produces the strongest exotoxin. Outside the body, it forms spores that are resistant to physical and chemical factors: they remain in the soil for up to 10 years, only die when boiled
  15. Crib. Infectious diseases, 2010
    Typhoid fever. Paratyphoid A and B. cholera. Amoebiasis. Yersiniosis. Ascaridosis. Trichinosis. Typhus fever, Brill-Zinsser batch. Echinococcosis. Enterobiosis. Q fever. Lime borreliosis. Leptospirosis. Tularemia. Anthrax. Meningococcal infection. Plague. Tetanus. Rabies. Ornithosis. Legionellosis. Erysipelas. Influenza. Food.
  16. Preventive planning
    Owners should not be deceived by the idea that horses will live their lives without any illnesses. It can be useful to make a disease prevention plan. Mark for yourself all the requirements that need to be met, what has already been done and what else needs to be done. Our memory is unreliable; Planning helps not to miss the important and do everything according to the rules. An example of such planning is a program.
  17. Rabies.
    Training task: using the algorithm, determine the clinical stage of rabies, conduct a differential diagnosis; prescribe treatment measures for emergency care and treatment at the hospital stage. Task for self-study. Using the textbook, Infectious Diseases Manual, lecture material and a list of recommended literature for acquiring the necessary basic
  18. Acute respiratory failure
    Acute respiratory failure complicates the course of many infectious diseases due to impaired pulmonary ventilation as a result of laryngospasm (tetanus, rabies) and acute inflammation of the larynx (diphtheria, viral croup), respiratory muscle paralysis (botulism, diphtheria), obstructing tracheobronchial and alveolar inflammatory edema (influenza, acute respiratory infections, measles
  19. BACTERIAL TOXINS
    Classification of bacterial toxins. In 1887, Louis Pasteur conducted experiments proving that the substances that are formed as a result of the vital activity of microorganisms and are in the nutrient broth can cause the same clinical signs of the disease as when infected with the pathogen itself. E. Ru and A. Yersin in their experiments of 1889 confirmed this conclusion.
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