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Pulmonary hypertension

580. REDUCE HYPERTENSION OF A SMALL CIRCLE

BLOOD CIRCULATION 1. HEPARIN 2. EUFILLIN 3.

NITROGLYCERINE 4. PREDISOLON 5. NORADRENALINE

1) only 1,2,4 is true

2) only 2.3 is true

3) only 1.4 is true

4) only 3 is true

5) everything is true



121







581. FOR PULMONARY HYPERTENSION VENOUS TYPE NOT

CHARACTERISTIC

1) pulmonary capillary pressure of 10 mm RT. Art.

2) cough, hemoptysis

3) congestive wheezing in the lungs

4) shortness of breath, asthma attacks

5) enhancement of vascular pattern during radiography



582. MORE THAN PRIMARY PULMONARY HYPERTENSION

IDENTIFIED IN THE AGE

1) in infancy

2) in early childhood

3) in adolescence

4) aged 20-40 years

5) in old age



583. In pathogenesis, LH ALLOCATE ALL

PATHOPHYSIOLOGICAL MECHANISMS, COMBINATION

WHICH THE SUMMARY OF THE PROCESSES DISCLOSES

SIMULATION OF PULMONARY VESSELS. NOT TO THEM

Is related

1) vasoconstriction.

2) reduction of the pulmonary vascular bed.

3) a decrease in the elasticity of the pulmonary vessels.

4) obliteration of the pulmonary vessels (thrombosis in situ,

smooth muscle cell proliferation)

5) vasodilation



584. STARTING FROM THE BASIC ASPECTS OF PATHOGENESIS, OBJECTIVES

PRIMARY PULMONARY HYPERTENSION THERAPY

1) vasodilation - relaxation of smooth muscle cells

pulmonary vessels

2) prevention or regression of remodeling

pulmonary vessels

3) chronic anticoagulation - prevention

pulmonary embolism and / or thrombosis in situ

4) all of the above

5) none of the above



585.
X-RAY SIGN OF PULMONARY HEART

IS AN

1) decreased transparency of the pulmonary fields

2) increased transparency of the pulmonary fields





122





3) the swelling of the second arc along the left contour in a straight line

projections

4) lengthening of the lower arc along the left contour in the second oblique

position

5) a shift of the right cardiovascular angle down by

direct radiograph



586. THE MOST FREQUENT REASON FOR THE DEVELOPMENT OF THE CHRONIC

PULMONARY HEART IS

1) chronic obstructive bronchitis

2) pulmonary tuberculosis

3) interstitial pulmonary fibrosis (syndrome

Hamman - Rich)

4) pulmonary embolism

5) kyphoscoliosis



587. TO INCREASE THE RIGHT VENTRICLE CHARACTERISTIC

1) “lifting” apical impulse

2) epigastric pulsation

3) echocardiographic size of the right ventricle (in

M-mode) = 20 mm

4) the shift of the border of relative cardiac dullness

up

5) deep tooth S in assignment V1



588. A SIGN OF INSUFFICIENCY OF LEGAL DEPARTMENTS

HEART IS

1) lowering blood pressure

2) increased central venous pressure (CVP)

3) decrease in CVP

4) anemia

5) polycythemia



589. IN THE RIGHT VENTRICULAR INSUFFICIENCY

DEVELOPING

1) systemic venous congestion

2) a decrease in central venous pressure (CVP)

3) arterial hypertension

4) pulmonary edema

5) vegetative-vascular crisis
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Pulmonary hypertension

  1. PRIMARY PULMONARY HYPERTENSION
    John Ross, Jr. Primary (or idiopathic) pulmonary hypertension is an infrequent disease, the diagnosis of which is based on the exclusion of other known and more common causes of increased pressure in the pulmonary circulation. In most cases, primary pulmonary hypertension is diagnosed in women aged 20-40 years, which, however, does not exclude
  2. CLASSIFICATION OF PULMONARY HYPERTENSIONS
    PRIMARY PULMONARY HYPERTENSION Primary pulmonary hypertension (PLH) is a disease of unclear etiology, the diagnostic criteria for which are the following symptoms according to M. Riedel and J. Widimsky (1987): 1. An increase in pulmonary trunk pressure and normal jamming pressure. 2. The absence of heart and lung diseases. 3. The absence of local anomalies of the pulmonary vessels according to
  3. Persistent pulmonary hypertension
    Persistent pulmonary hypertension (PLH) in newborns is a disease characterized by severe arterial hypoxemia resulting from an increase in pulmonary vascular resistance with bypass blood from the pulmonary arteries into the systemic circulation. This pathology is also known as persistent fetal circulation syndrome. Etiology. PLG often complicates the course of such
  4. Persistent pulmonary hypertension in newborns
    SYNONYMS Persistent pulmonary hypertension in newborns, persistent fetal circulation. DEFINITION Persistent fetal blood circulation in a newborn (SPSF) is a symptom complex characterized by refractory arterial hypoxemia resulting from persistent high pulmonary vascular resistance and bypass blood flow from right to left through the OA and oval window. CODE
  5. Primary pulmonary hypertension (AERSA ​​syndrome)
    It is characterized by hypertrophy of the myocardium of the right ventricle, expansion of the trunk of the pulmonary artery. Morphologically revealed fibrosis and fibroelastosis of intima, fibrinoid-necrotic arteritis of small branches of the pulmonary artery and thrombosis. It is believed that the immediate cause is fibrosis and fibroelastosis of the muscle layer of the pulmonary arterioles, most likely associated with genetically determined
  6. MODERN ASPECTS OF DIAGNOSIS AND TREATMENT OF PULMONARY HYPERTENSION
    Pulmonary hypertension (LH) is a pathological syndrome caused by an increase in blood pressure in the pulmonary circulation (MCC). The pressure in the ICC is considered increased if it exceeds normal values: systolic - 26-30 mm Hg, diastolic 13-19 mm Hg LH causes severe disturbances in vital organs and systems, especially in the lungs and heart. It causes
  7. PULMONARY HYPERTENSION
    PULMONARY
  8. Pulmonary heart and pulmonary circulation disorders
    ICD code: (126-128) 126 Pulmonary embolism 126.0 Pulmonary embolism with reference to acute pulmonary heart 126.9 Pulmonary embolism without mention of acute pulmonary heart 127 Other forms of pulmonary heart failure 127.0 Primary pulmonary hypertension 127.1 Kyphoscoliotic heart disease 127.8 Other specified forms of pulmonary heart failure 127.9
  9. 52. LUNG HEART. ETHIOLOGY, PATHOGENESIS OF ACUTE AND SUBCUTANEOUS, CHRONIC PULMONARY HEART, CLINIC, DIAGNOSTIC, TREATMENT PRINCIPLES.
    Pulmonary sero-pathological condition, characterized by hypertrophy of the right ventricle caused by hypertension of the pulmonary circulation, which develops with damage to the bronchopulmonary apparatus, pulmonary vessels, chest deformity, or other diseases that impair lung function. Acute heart lay-wedge symptom complex arising from pulmonary artery thromboembolism, and with
  10. Hypertension
    In the broad sense of the word, the term "hypertension" means an increase in hydrostatic pressure in the vessels, hollow organs or cavities of the body. However, in this section we will focus only on vascular disease, the main clinical sign of which is a steady increase in blood pressure - arterial hypertension. The main target for this pathology are arteries
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