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Dyscirculatory encephalopathy

Slowly progressing insufficiency of blood supply to the brain, leading to increasing diffuse structural changes with impaired brain function. It manifests itself as a multi-focal disorder of brain functions. The initial symptoms appear in the form of asthenia, which often makes it difficult to make a diagnosis at this stage of the development of the disease, since such symptoms often accompany the initial stage of mental disorders, the prodromal phase of some infectious diseases, as well as a signal of the presence of somatic diseases or banal overwork. Therefore, the diagnosis of “discirculatory encephalopathy” is made on the basis of the prolonged (more than 3 months) presence of the main symptoms (dizziness, impaired memory, speech, increased fatigue, impaired coordination, etc.).
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Dyscirculatory encephalopathy

  1. Metabolic encephalopathy
    (Internet) Encephalopathy (encephalopathia, anat. encephalon brain + Greek pathos suffering, illness; synonym: pseudoencephalitis, cerebropathy, encephalosis) - diffuse brain damage syndrome, which is based on the mechanism of ischemia (impaired blood supply), hypoxia (oxygen starvation ) brain tissue and, as a consequence, the phenomena of death of nerve cells due to
  2. Perinatal encephalopathy
    A.A.Savina and Yu.K. Khokhlov (1996) believe that, taking into account the so-called perinatal and early postnatal encephalopathies, the number of children with pathology of the central nervous system increases even more. According to I.V. Nezgovorova et al. (1994), 76% of newborns suffer perinatal encephalopathy of various origins, and in 24% it is diagnosed late and leads to development
  3. HEPATIC ENCEPHALOPATHY
    Hepatic encephalopathy (hepatathy) is a neuropsychiatric syndrome that can develop with severe liver failure, intoxication, sepsis, severe infections due to liver dysfunction or due to portosystemic shunts. Despite active research, the exact pathogenesis of hepatic encephalopathy is still not disclosed. It has been shown that many neurotoxins and
  4. Toxic encephalopathy
    Protocol code: 01-012в Profile: pediatric Stage: hospital Purpose of stage: 1. stopping the threat of violation of vital functions (restoration of consciousness, cardiac activity); 2. prevention of complications; 3. stabilization of the state; 4. relief of seizures; Duration of treatment (days): 10 ICD code: G 92 Toxic encephalopathy Definition: Toxic encephalopathy - represents
  5. Hypertensive (dyscirculatory) encephalopathy
    Hypertensive encephalopathy is a diffuse violation of the functions of the brain caused by its edema as a result of the loss of the ability of the central nervous system (CNS) to maintain vascular autoregulation. The rate at which blood pressure rises is likely to be as important as its absolute level. In chronic arterial hypertension, changes in cerebral autoregulation are directed
  6. Encephalopathy of Mink
    Mink encephalopathy (vector-borne mink encephalopathy) is a prion disease characterized by progressive damage to the central nervous system, degenerative changes in the brain and death of animals. Historical background, distribution, hazard and damage. The disease was first discovered in 1947, and then after a long break again in 1961 in a number of farms in the USA and Canada.
  7. Metabolic encephalopathy
    The term "encephalopathy" is used to refer to diffuse disorders of the brain due to impaired functions of many neurons in various parts of the central nervous system. In most cases, encephalopathy occurs when there is a disturbance in the processes of oxidative metabolism observed with hypoxia or global brain ischemia. Hypoxic brain damage. For the human brain
  8. Spongioform encephalopathies
    Etiology. It is suggested that spongioform encephalopathies (syn: slow prion infections, Creutzfeldt-Jakob disease and kuru) are caused by a protein-like particle - prion. These particles have a molecular weight of 60–100 kDa. Kuru is endemic to the islands of Papua New Guinea and is currently extremely rare. Creutzfeldt – Jakob disease was described in 1921 and is currently not uncommon
  9. Bovine spongiform (spongioform) encephalopathy
    Bovine spongiform (spongioform) encephalopathy (BSE) is a slowly developing disease with damage to the central nervous system related to prion (slow) infections. Etiology. A specific feature of the causative agent of GE cattle is its very small size (m 28-30 KD) and high resistance to chemical and physical factors. PrP27-32 proteins in brain tissue and spleen
  10. Toxic encephalopathy treatment
    Coma Specific antidote therapy is one of the main methods of treating coma in acute poisoning. Adequate detoxification and antidote therapy, timely correction of hypoxia and its consequences is important in the treatment of toxic coma. Toxic cerebral edema The most formidable complication of toxic encephalopathy is the development of toxic cerebral edema.
  11. Acute hypertensive encephalopathy
    Acute hypertonic encephalopathy is a syndrome resulting from a rapid sharp rise in blood pressure in patients with arterial hypertension of various origins. In this case, multiple small foci of hemorrhages and ischemia and cerebral edema may occur. D - ka: The clinical picture is characterized, first of all, by pronounced cerebral symptoms - a rapidly growing headache
  12. Acute hypertensive encephalopathy
    This disease refers to late hypertensive brain lesions and is manifested in acute diffuse cerebrovascular accident with rapidly developing syndrome of increased intracranial pressure and meningeal syndrome. Sometimes it occurs in the transitional phase of hypertension, but much more often manifests itself in later stages. Most authors link
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