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Post-hemorrhagic anemia

Acute post-hemorrhagic anemia

Develops as a result of a single rapid massive blood loss.

Violations are due to a decrease in circulating blood volume and hypoxia. In response to blood loss, compensation mechanisms are included, aimed at restoring lost blood volume and fighting anemic condition (hypoxia).

A picture of blood. On 1 - 2 days after blood loss, normochromic anemia develops. On the 4th - 5th day, reticulocytosis occurs, polychromatophils, polychromatophilic and oxyphilic normoblasts (normocytes) appear. Due to the insufficient hemoglobinization of erythrocytes, anemia becomes hypochromic in nature, accompanied by moderate neutrophilic leukocytosis with a shift to the left of the regenerative type.

Chronic post-hemorrhagic anemia

It develops as a result of repeated small blood loss.

The pathogenesis is iron deficient (see below).
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Post-hemorrhagic anemia

  1. Anemia due to blood loss, post-hemorrhagic anemia
    Anemia due to blood loss, post-haemorrhagic
  2. Post-haemorrhagic anemia
    Causes of acute anemia from blood loss are various external injuries, accompanied by damage to blood vessels or bleeding from internal organs. The picture of acute post-hemorrhagic anemia immediately after the bleeding consists of the actual anemic symptoms and the collapse picture. There may be a sharp paleness of the skin, dizziness, fainting,
  3. Post-hemorrhagic anemia
    CLASSIFICATION According to the time of occurrence and pathogenesis post-hemorrhagic anemia can be divided into 3 main groups. • Obstetric reasons. o Premature placental abruption. o Dissection of the placenta at caesarean section. o Cord breaking. o umbilical cord hematoma. o Rupture of abnormal vessels. • “Internal” causes. o Feto-maternal losses. - Malformation of the placenta. - Chorioangioma and
  4. Anemia Acute post-hemorrhagic anemia (code D 62)
    Definition Clinical symptom complex caused by massive single blood loss, characterized by hypovolemia and normochromic anemia. Etiology, pathogenesis. A one-time loss of more than 500 ml of blood due to internal or external bleeding leads to hypovolemia with the subsequent development of normochromic anemia, stimulation of hemopoiesis and restoration of the initial parameters of peripheral
  5. Anemia associated with blood loss (post-hemorrhagic)
    Anemia associated with blood loss
  6. CHRONIC POST-HEMORRHAGIC ANEMIA
    Chronic post-hemorrhagic anemia develops as a result of either a single, but abundant blood loss, or minor, but prolonged repeated blood loss (Fig. 38). Most often chronic post-hemorrhagic anemia is observed when bleeding from the gastrointestinal tract (ulcer, cancer, hemorrhoids), renal, uterine. Even occult blood loss with ulcers or neoplastic
  7. Chronic post-hemorrhagic anemia
    It develops as a result of long-lasting non-abundant bleeding. Chronic post-hemorrhagic anemia in children is the main cause of iron deficiency. The following pathological conditions can be the causes of repeated small blood loss: 1. Diseases of the gastrointestinal tract: erosive and ulcerative lesions of the gastrointestinal tract, polyposis, diaphragmatic hernia, mucous membrane duplication
  8. Acute post-hemorrhagic anemia
    This condition develops as a result of a single massive blood loss. The reasons leading to the development of acute post-hemorrhagic anemia in children of different age groups are presented in Table. 12. Clinical picture Small blood loss in older children does not cause marked clinical symptoms and is relatively well tolerated. Significant blood loss to children is tolerated.
  9. ANEMIA DUE TO TOXIC EXPOSURE OF THE BONE MARROW (MYETHOXICAL ANEMIA)
    THYROORTHEUS ANEMIA Thyroid stimulation anemia is anemia that develops on the basis of hypothyroidism. Experimental work confirms the important role of thyroid hormone - thyroxin - in stimulating normal blood formation. Animals deprived of the thyroid gland, quickly anemiziruyutsya, the introduction of the same thyroid drugs to thyroidectomized animals cures them from the effects of thyroid-induced
  10. Acute Post-Hemorrhagic Anemia
    Causes of acute anemia from blood loss are various external injuries (injuries), accompanied by damage to blood vessels, or bleeding from internal organs. Gastrointestinal bleeding, bleeding into the abdominal cavity (rupture of the fallopian tube in ectopic pregnancy), renal, pulmonary (tuberculosis, abscess), uterine, and also bleeding are most commonly observed.
  11. ANEMIA UNDER CONDITIONS OF FUNCTIONAL AGASTRY (AGASTRAL ANEMIA)
    In contrast to the actual agastric anemia associated with partial or complete anatomical removal of the stomach, we distinguish anemia that develops in conditions of functional agastria, when the anatomically preserved stomach partially or completely turns off from the physiological process of digestion and, in particular, loses the ability to assimilate the external antianemic factor - Vitamin B12.
  12. Anemia associated with impaired synthesis of DNA and RNA (megaloblastic anemia)
    Megaloblastic anemia unites a group of acquired and hereditary anemias, a common feature of which is the presence of megaloblasts in the bone marrow. Regardless of the etiology of the patients, hyperchromic anemia with characteristic changes in the erythrocyte morphology is revealed - oval red blood cells, large (up to 12-14 microns or more). There are red blood cells with basophilic cytoplasm, in many of
  13. Anemia associated with impaired synthesis or utilization of porphyrins (sideroachrestic, sideroblastic anemia)
    This is a heterogeneous group of diseases, hereditary and acquired, associated with a violation of the activity of enzymes involved in the synthesis of porphyrins and heme. The term "sideroahresticheskaya anemia" was introduced by Iewer (1957). In case of sideroachrestric anemia, serum iron levels are elevated. In the bone marrow detect ring-shaped sideroblasts - nucleated red blood cells with a nucleolus corolla,
  14. Anemia due to bone marrow drainage, hypothyroidism, and aplastic anemia. MYELOPLASTIC SYNDROME
    Despite the large number of studies devoted to the study of hypo-and aplastic anemias, it is still not possible to create a rational classification of these conditions, since not only the issues of pathomorphogenesis, but even the definition of the very concept of "hypo (a) plastic anemia" is debatable. In accordance with modern concepts of the genesis of hypo- and
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