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Markers of inflammation


CRP (C-reactive protein) produced by the liver is a marker of inflammation. Increased CRP content means that the inflammatory system is in the on state, which threatens not only heart disease, but also other disorders associated with inflammation. Find out with the doctor the cause of this condition. What is it: a nutritional deficiency, a latent infection (parasitosis) or something else? Your investigation will give invaluable results. Other markers of inflammation: ESR (erythrocyte sedimentation rate), insulin levels in the blood (insulin is a hormone that promotes inflammation) and fibrinogen (systems of inflammation and blood coagulation use the same molecules).
AA / EPA1 RATIO
This systemic inflammation marker is more complex than the CRP assay and detects inflammation at a more subtle level. It shows the presence of latent inflammation. The higher the ratio, the more intense the latent inflammation.
If it exceeds 10, then the body has an inflammatory process. A good ratio is 3, ideal is 1.5. In the average American, it is 11, and for those who have already developed inflammatory diseases, it can reach 20. A high ratio indicates that you are rapidly aging and losing health.
1 Arachidonic acid / eicosapentaenoic acid is actually the ratio of omega-6 and omega-3 fatty acids. - Approx. ed.
Remember that inflammation has a protective function in the body. The problem occurs when it develops for no apparent reason, due to the imbalance between substances that promote inflammation and interfere with it. Too many inflammatory substances are just as bad as too few. If the AA / EPA ratio is too low — for example, 0.7 — you will be more susceptible to infection, and you may not develop the necessary inflammatory response.
LIPOPROTEIN (A)
This type of fat is considered even more harmful than the so-called bad cholesterol, and increases the probability of developing coronary heart disease by 7 times. It does not affect any statins or exercise. Niacin helps, but not always. Finding elevated lipoprotein levels in my patients, I prescribe a computerized angiogram for them.
URIC ACID
Toxic by-product of animal protein processing that causes gout (inflammation in the joints) and eats away the walls of the arteries. Increases plaque formation in arteries.
VITAMIN D
There is growing evidence of a link between vitamin D deficiency and cardiovascular diseases, as well as depression, osteoporosis, and cancer. Today, people are so zealously protected from the sun that they constantly lack this vitamin. This entails disastrous consequences. Early detection of low levels of vitamin D should be a signal for you to change your diet, add supplements to it and increase your time in the sun.
HOMOCYSTEIN
Amino acid, a byproduct of protein processing.
It becomes toxic if the liver does not effectively remove it. High levels of homocysteine ​​in the blood plasma creates a predisposition to coronary heart disease, Alzheimer's disease, premature birth and other disorders of the reproductive system in young women. In this case, you should conduct a detoxification program and supplement the diet with supplements with vitamin B.
FUNCTION OF THE THYROID GLAND
Most doctors prescribe tests for T ^ (a hormone that stimulates the thyroid gland) and T4 (thyroxin), but it is also necessary to determine the content of free T3 - the active thyroid hormone. To transform T4 into T3, and then into an active form - free T3 - the body needs certain vitamins and minerals that help strengthen the thyroid gland and metabolism.
Antibodies to thyroglobulin. Autoimmune processes caused by gluten can lead to the formation of antibodies to proteins of the thyroid system, called thyroglobulins. The timely detection of their increase contributes to the prevention of serious problems in the future.
Damage to the inner walls of the intestine allows access to the intestinal lymphoid tissue antigens, which normally should be filtered out. Many allergic reactions begin this way. Gluten, a protein found in wheat and other grains, can trigger an immune response called celiac disease. In severe form, it can be fatal.
Iodine content. The thyroid gland uses iodine to produce its hormones. Our food contains insufficient amounts of it. When iodine deficiency is large, gout develops, but lately there has been increasing evidence of a connection between a small iodine deficiency and various diseases, including cardiovascular diseases.
If you have impaired thyroid function or symptoms that you suspect are related to abnormalities in its work, ask your doctor to prescribe an assay for iodine absorption.
MERCURES AND OTHER HEAVY METALS
Mercury is called a “great imitator” because it causes many different diseases: psychiatric disorders, cancer, autoimmune disorders. If your diagnosis is unclear, and the symptoms, despite all efforts, do not go away or you suspect that you have been exposed to heavy metals (have eaten a lot of tuna or other fish contaminated with mercury, or you have silver amalgam fillings in your mouth) Give you a test for mercury and heavy metals. The mercury content in the blood and hair is a useful indicator, but it does not determine the extent to which the body is affected by mercury. The only reliable analysis in this regard is the analysis of daily urine using the chelating substance DMSA.
ANALYSIS OF ORGANIC ACIDS
This analysis makes it possible to determine the mode of supplementation that is most suitable for you, instead of taking those that are advertised at the moment (they may or may not be suitable for you).
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Markers of inflammation

  1. INFLAMMATION: DEFINITION, ESSENCE, BIOLOGICAL SIGNIFICANCE. MEDIATORS OF INFLAMMATION. LOCAL AND GENERAL INFLAMMATION. ACUTE INFLAMMATION: ETIOLOGY, PATHOGENESIS. MORPHOLOGICAL MANIFESTATION OF EXCUTIVE INFLAMMATION. THE OUTCOMES OF ACUTE INFLAMMATION
    Inflammation is a biological, and at the same time, a key general pathological process, the expediency of which is determined by its protective-adaptive function, aimed at eliminating the damaging agent and restoring damaged tissue. In medicine, to indicate inflammation to the name of the organ in which the inflammatory process develops, add the ending "it" - myocarditis, bronchitis,
  2. Inflammation. Definition, essence, mediators of inflammation. Local and general manifestations of exudative inflammation, morphological manifestations of exudative inflammation. The answer is the acute phase. Ulcerative-necrotic reactions in inflammation.
    1. The main processes that develop in the body in response to tissue damage are 1. amyloidosis 2. inflammation 3. regeneration 4. formation of granulomas 5. hyperplasia of cell ultrastructures 2. Inflammation is 1. hyperplasia of cell ultrastructures 2. restoration of lost structures 3 Unrestrained growth of cellular elements 4. exudative-proliferative response to damage 5. cellular
  3. Productive and chronic inflammation. Granulomatosis The morphology of specific and non-specific inflammation.
    1. Chronic inflammation manifests itself by a simultaneous combination of 1. inadequate repair 2. angiogenesis, scarring 3. reactive changes 4. tissue damage 5. embolism 2. Causes of chronic inflammation 1. acute infection 2. persistent infection 3. prolonged exposure to toxic substances 3. Chronic inflammation characterized by 1. amyloid deposition 2. mononuclear infiltration
  4. Biomolecular markers of tumors.
    Traditional morphological studies, as a rule, allow to make an accurate diagnosis in cases of differentiated tumors and their metastases. In cases of poorly differentiated and undifferentiated malignant tumors in the diagnosis, modern research methods are used to diagnose changes at the ultrastructural and molecular genetic levels. To this end
  5. Course of inflammation. Acute and chronic inflammation
    The course of inflammation is determined by the body's reactivity, type, strength, and duration of action of the phlogogen. There are acute, subacute and chronic inflammation. Acute inflammation is characterized by: - ​​intense and relatively short (usually 1-2, up to a maximum of 4-6 weeks) duration (depending on the damaged organ or tissue, the extent and scale of their alteration,
  6. General information about inflammation acute inflammation
    Inflammation is a complex, local and general pathological process that occurs in response to damage or the action of a pathogenic stimulus and manifests itself in reactions aimed at eliminating damage products, and, if possible, agents (stimuli), as well as resulting in maximum recovery for these conditions. in the damage zone. Inflammation, has a protective and adaptive
  7. Proliferation and outcomes of inflammation. Types of inflammation
    Inflammation always begins with damage and cell death. But at a certain stage, when the processes of repairing damage, cleansing from the entire dead, alien to the body, take effect, infiltration, suppuration and the associated processes of proteolysis and necrosis stop and recovery processes come to the fore. In accordance with this, the cellular composition of the inflammatory
  8. Nutrition during exacerbation of liver inflammation and acute inflammation of the gallbladder
    Nutrition is recommended for patients with acute cholecystitis and hepatitis, chronic cholecystitis and hepatitis, cirrhosis of the liver with moderately severe liver failure, gallstone disease, and also with simultaneous damage to the liver and biliary tract, stomach and intestines. Food is cooked on water or steamed, wiped. Excluded are products that enhance the fermentation and rotting processes in
  9. Recommended products for exacerbation of liver inflammation and acute inflammation of the gallbladder
    Recommended: yesterday's wheat bread; soups are cooked on mucous broth with grated cereals, vegetables or on vegetable broths with finely chopped vegetables - potatoes, carrots, zucchini, pumpkin, boiled cereals - rice, semolina, oatmeal cereals, noodles, soups can be added egg-milk mixture, which is prepared a mix of raw eggs with an equal volume of milk, and fill
  10. Biological markers of bronchial asthma
    With reference to bronchial asthma, such indicators are morphological and functional changes in eosinophil. Recently, great importance is attached to the level of nitric oxide (NO) in exhaled air as a biomarker. The source of nitric oxide is involved in the inflammatory process of epithelial cells. Measurements of the fraction of exhaled NO (FeNO) may be useful for
  11. Features of the development of the inflammatory reaction, depending on the localization of inflammation, the reactivity of the organism, the nature of the etiological factor. The role of age in the development of inflammation
    Regardless of the localization of the inflammatory process and the origin of the etiological factor, a standard complex of vascular and tissue changes always occurs in the zone of acute inflammation. The reaction of the tissues to the action of the damaging factor is of a phase nature and is manifested by alteration, exudation and proliferation. Simultaneously with tissue disorders, a complex of vascular arises
  12. Electrophysiological markers of sympathetic catecholamine aggression
    According to the personal experience of the author of this abstract review, some electroencephalographic phenomena can be electrophysiological expression and a marker of sympathetic catecholamine aggression. For example, hypersynchronous beta-1 oscillations may be due to the inhibition by sympathetic catecholamines of the GABAergic inhibitory interneurons of layer III of the cortex, followed as spontaneous
  13. ASSOCIATION OF A2 ALLE MICROSATELLITE MARKER TNFA WITH SOME HISTOLOGICAL CHARACTERISTICS OF BC
    Malivanova T.F., Anurova O.A., Yurchenko V.A., Mazurenko N.N. Russian Cancer Scientific Center tumor and lobular histological type of breast cancer. Task
  14. PECULIARITIES OF EXPRESSION OF CYTOSTATIC SENSITIVITY MARKERS IN PATIENTS WITH DIFFERENT SOLID TUMORS
    Zhabina A.S.1, Protsenko S.A.1, Ievleva A.G.1, Teletaeva G.M., Moiseenko V.M.2, Imyanitov E.N.1 1FGU Research Institute of Oncology named after N.N. Petrov Rosmedtechnology; 2 UO DPO SPbMAPO Roszdrav, St. Petersburg Research Objectives To assess the expression of markers of sensitivity to chemotherapy in patients with various solid tumors. Research methods Paraffin blocks are included in the work.
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