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Markers of inflammation


CRP (C-reactive protein), produced by the liver, is a marker of inflammation. Increased CRP means that the inflammatory system is in the switched on state, which threatens not only heart disease, but also other disorders associated with inflammation. Find out with the doctor the cause of this condition. What is it: deficiency of nutrients, latent infection (parasitosis) or something else? Your investigation will yield invaluable results. Other markers of inflammation: ESR (erythrocyte sedimentation rate), insulin level in the blood (insulin is a hormone that promotes inflammation) and fibrinogen (the systems of inflammation and clotting use the same molecules).
RATIO OF AA / EPA1
This marker of systemic inflammation is more difficult than analysis for CRP and reveals inflammation at a more subtle level. It shows the presence of latent inflammation. The higher the ratio, the more intense the latent inflammation.
If it exceeds 10, then in the body there is an inflammatory process. A good ratio is 3, ideal is 1.5. In an average American, it is 11, and those who have already developed inflammatory diseases, can reach 20. A high ratio indicates that you quickly grow old and lose health.
1 Arachidonic acid / eicosapentaenoic acid is actually the ratio of omega-6 and omega-3 fatty acids. - Note. Ed.
Remember that inflammation performs a protective function in the body. The problem arises when it develops for no apparent reason, due to the imbalance between substances that contribute to inflammation and prevent it. Too much inflammation-preventing substance is as bad as too little. If the AA / EPA ratio is too low - for example, 0.7, - you will be more exposed to infections, and you may not develop the necessary inflammatory response.
LIPOPROTEIN (A)
This type of fat is considered even more harmful than the so-called bad cholesterol, and 7 times increases the likelihood of developing coronary heart disease. It is not affected by statins or exercise. Niacin helps, but not always. Detecting my patients with an elevated level of lipoprotein, I assign them a computer angiogram.
URIC ACID
A toxic byproduct of animal protein processing that causes gout (inflammation in the joints) and corrodes the walls of the arteries. Increases the likelihood of plaque formation in the arteries.
VITAMIN D
There is increasing evidence of a link between vitamin D deficiency and cardiovascular disease, as well as depression, osteoporosis and cancer. Today, people are so zealously protected from the sun that they constantly lack this vitamin. This entails catastrophic consequences. Early detection of a low level of vitamin D should be a signal for you to change the diet, add supplements to it and increase the time spent in the sun.
HOMOCYSTEINE
Amino acid, a byproduct of protein processing.
It becomes toxic if the liver does not display it effectively. The high content of homocysteine ​​in the blood plasma creates a predisposition to coronary heart disease, Alzheimer's disease, premature birth and other reproductive system disorders in young women. In this case, you should conduct a detoxification program and supplement the diet with supplements with vitamin B.
FUNCTION OF THE THYROID GLAND
Most doctors prescribe tests for T ^ (a hormone that stimulates the thyroid gland) and T4 (thyroxine), but it is also necessary to determine the content of free T3 - the active hormone of the thyroid gland. To convert T4 to T3, and then into the active form - free T3 - the body needs certain vitamins and minerals that contribute to the strengthening of the thyroid gland and metabolism.
Antibodies to thyroglobulins. Autoimmune processes caused by gluten can lead to the formation of antibodies to proteins of the thyroid system called thyreoglobulins. Timely detection of their increase helps to prevent serious problems in the future.
Damage to the inner walls of the intestine opens access to the intestinal lymphoid tissue antigens, which normally should be filtered out. Similarly, many allergic reactions begin. Gluten, a protein found in wheat and other grains, can cause an immune reaction called celiac disease. In severe form, it can lead to death.
The content of iodine. The thyroid gland uses iodine to produce its hormones. Our food contains insufficient quantity. When iodine deficiency is great, gout develops, but lately there is increasing evidence of a link between a small deficiency of iodine and various diseases, including cardiovascular.
If your thyroid function is impaired or if you have symptoms that you suspect are related to violations in her work, ask your doctor to give you an analysis for iodine digestion.
MERCURY AND OTHER HEAVY METALS
Mercury is called a "great imitator" because it causes a variety of diseases: psychiatric disorders, cancer, autoimmune disorders. If your diagnosis is unclear and the symptoms do not go away, or you suspect that you have been exposed to heavy metals (ate a lot of tuna or other fish contaminated with mercury, or you have silver amalgam fillings in your mouth), ask your doctor assign you an analysis for the presence of mercury and heavy metals. The content of mercury in blood and hair is a useful indicator, but it does not determine to what extent the organism is affected by mercury. The only reliable analysis in this regard is the analysis of 24-hour urine using the DMSA chelate.
ANALYSIS OF ORGANIC ACIDS
This analysis makes it possible to determine the most suitable for you mode of reception of additives, instead of accepting those that are advertised at the moment (they may or may not be suitable for you).
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Markers of inflammation

  1. INFLAMMATION: DEFINITION, ESSENCE, BIOLOGICAL VALUE. MEDIATORS OF INFLAMMATION. LOCAL AND GENERAL STAGES OF INFLAMMATION. ACUTE INFLAMMATION: ETIOLOGY, PATHOGENESIS. MORPHOLOGICAL STATEMENT OF EXODUSIVE INFLAMMATION. OUTCOME OF ACUTE INFLAMMATION
    Inflammation is a biological, and at the same time, key all-pathological process, the expediency of which is determined by its protective-adaptive function aimed at eliminating the damaging agent and restoring the damaged tissue. In medicine, to mark inflammation to the name of the organ in which the inflammatory process develops, add the ending "it" - myocarditis, bronchitis,
  2. Inflammation. Definition, essence, mediators of inflammation. Local and general manifestations of exudative inflammation, morphological manifestations of exudative inflammation. The acute phase response. Ulcerative necrotic reactions during inflammation.
    1. The main processes that develop in the body in response to tissue damage are 1. amyloidosis 2. inflammation 3. regeneration 4. formation of granulomas 5. hyperplasia of ultrastructure of cells 2. Inflammation is 1. hyperplasia of ultrastructure of cells 2. restoration of lost structures 3 unrestrained growth of cellular elements 4. exudative-proliferative reaction to damage 5. cellular
  3. Productive and chronic inflammation. Granulomatosis. Morphology of specific and nonspecific inflammation.
    1. Chronic inflammation is manifested by a simultaneous combination of 1. Inadequate repair 2. Angiogenesis, scarring 3. Reactive changes 4. Damage to tissues 5. Embolism 2. Causes of chronic inflammation 1. Acute infection 2. Persistent infection 3. Prolonged exposure to toxic substances 3. Chronic inflammation characterized by 1. deposition of amyloid 2. mononuclear infiltration
  4. Biomolecular markers of tumors.
    Traditional morphological studies, as a rule, allow an accurate diagnosis in cases of differentiated tumors and their metastases. In cases of low-grade and undifferentiated malignant tumors, modern diagnostic methods are used to diagnose changes at the ultrastructural and molecular-genetic levels. To this end
  5. The course of inflammation. Acute and chronic inflammation
    The course of inflammation is determined by the reactivity of the organism, the type, strength, and duration of the action of the phlogogen. There are acute, subacute and chronic inflammation. Acute inflammation is characterized by: - ​​intensive course and relatively small (usually 1-2, maximum up to 4-6 weeks) duration (depending on the damaged organ or tissue, degree and extent of their alteration,
  6. General Information on Inflammation of Acute Inflammation
    Inflammation is a complex, local and general pathological process that arises in response to damage or the action of a pathogenic stimulus and manifests itself in reactions aimed at eliminating the damage products, and if possible also the agents (stimuli), and also resulting in the maximum recovery for these conditions in the area of ​​damage. Inflammation, has a protective-adaptive
  7. Proliferation and outcomes of inflammation. Types of inflammation
    Inflammation always begins with damage and death of cells. But at a certain stage, when the processes of elimination of damage, cleansing from all the deceased, alien to the body enter into force, infiltration, suppuration and the associated processes of proteolysis and necrosis are suspended and recovery processes come to the fore. In accordance with this, the cellular composition of the inflammatory
  8. Nutrition with exacerbation of liver inflammation and acute inflammation of the gallbladder
    Nutrition is recommended for patients with acute cholecystitis and hepatitis, chronic cholecystitis and hepatitis, cirrhosis of the liver with moderately expressed hepatic insufficiency, cholelithiasis, as well as with simultaneous lesion of the liver and bile ducts, stomach and intestines. The food is cooked on water or steam, rubbed. Products that enhance the processes of fermentation and putrefaction in
  9. Recommended products for exacerbation of inflammation of the liver and acute inflammation of the gallbladder
    Recommended: yesterday's wheat bread; soups are cooked on mucous broth with mashed cereals, vegetables or on vegetable broths with finely chopped vegetables - potatoes, carrots, zucchini, pumpkin, boiled rice, manna, oatmeal, noodles, you can add egg-milk mixture to soups, which is prepared the connection of a raw egg with an equal volume of milk, and fill
  10. Biological markers of bronchial asthma
    With respect to bronchial asthma, such indicators are the morphological and functional changes in eosinophil. Of great importance in recent times is the level of nitric oxide (NO) in the exhaled air as a biomarker. The source of nitric oxide is the epithelial cells involved in the inflammatory process. Measurements of the fraction of exhaled NO (FeNO) may be useful for
  11. Features of the development of the inflammatory reaction, depending on the localization of inflammation, the reactivity of the organism, the nature of the etiologic factor. The role of age in the development of inflammation
    Regardless of the localization of the inflammatory process and the origin of the etiological factor in the acute inflammation zone, a standard set of vascular and tissue changes always occurs. The reaction from tissues to the effect of the damaging factor is phase-like and manifests itself by alteration, exudation and proliferation. Simultaneously with tissue disorders, a complex of vascular
  12. Electrophysiological markers of sympathetic catecholamine aggression
    According to the personal experience of the author of this abstract review, some electroencephalographic phenomena may be an electrophysiological expression and a marker of sympathetic catecholamine aggression. For example, hypersynchronous beta-1 oscillations may be due to inhibition by sympathetic catecholamines of GABA-ergic inhibitory interneurons of the third layer of the cortex with subsequent as spontaneous
  13. ASSOCIATION a2 OF ALLEY OF MICROSATELLITE MARKER TNFa WITH CERTAIN HISTOLOGICAL CHARACTERISTICS OF BC
    Malivanova TF, Anurova OA, Yurchenko VA, Mazurenko NN RNTS NN Blokhin RAMS, Moscow Research objective: We previously found the association of some alleles of the polymorphic microsatellite marker TNFa and point replacement in the promoter region of the tumor necrosis factor -308 gene (G> A) TNF with the receptor-negative status tumor and lobular histological type of breast cancer. The task
  14. PECULIARITIES OF EXPRESSION OF MARKERS OF SENSITIVITY TO CYSTOSTASTICS IN PATIENTS WITH VARIOUS SOLID TUMORS
    Zhabina A.S. 1, Protsenko S.A.1, Ievleva A.G.1, Teletaeva GM, Moiseenko V.M., 2, N.N.Ninitenov 1 1GNU of the Petrova Research Institute of Oncology Rosmedtechnologies; 2GUU DPO SPbMAPO Roszdrava, St. Petersburg Objectives of the study To evaluate the expression of markers of sensitivity to chemotherapy in patients with different solid tumors. Methods of research Paraffin blocks are included in the work
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