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T O M O R O R E N I

At present, the classification of a cold injury is as follows:

1. Acute defeat by cold:

a) freezing (general defeat);

b) frostbite (local defeat).

2. Chronic cold lesions:

a) chastity ("chronic frostbite");

b) cold neurovasculitis (trench stop, impression stop and a number of other synonyms).

EPIDEMIOLOGY

Frostbites are an important problem of surgery in a peaceful, and even more so in wartime. In the Soviet Army during the Great Patriotic War, sanitary losses from frostbite on separate fronts reached 3%, and in the navy - up to 5.4% of the total number of losses.

In peacetime, the extent of cold damage is not so significant. However, in





in northern regions of our country, they account for up to 30% of all cases of thermal injury, or 0.3-1% of all accidents, and 15 to 40% of the victims remain disabled.
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T O M O R O R E N I

  1. Vizer VA .. Lectures on therapy, 2011
    On the topic - almost completely cover the difficulties in the course of hospital therapy, the issues of diagnosis, treatment, in the presentation, laconic and quite affordable. Allergic diseases of the lungs Joint diseases Reiter's disease Sjogren's disease Bronchial asthma Bronchoectatic disease Hypertonic disease Glomerulonephrosfamiditis Herniasis of the esophagus Destructive lung diseases
  2. ALLERGIC DISEASES OF LUNGS
    In recent decades, there has been a significant increase in the number. patients with allergic diseases of the bronchopulmonary apparatus. Allergic lung diseases include exogenous allergic alveolitis, pulmonary eosinophilia, drug
  3. EXOGENOUS ALLERGIC ALVEOLITES
    Exogenous allergic alveolitis (syn: hypersensitive pneumonitis, interstitial granulomatous alveolitis) is a group of diseases caused by intensive and, more rarely, prolonged inhalation of antigens of organic and inorganic dusts and characterized by a diffuse, unlike pulmonary eosinophilia, lesion of alveolar and interstitial lung structures. The emergence of this group
  4. TREATMENT
    1. General measures aimed at dissociation of the patient from the source of antigen: compliance with sanitary and hygienic requirements in the workplace, technological improvement of industrial and agricultural production, rational employment of patients. 2. Drug treatment. In the acute stage - prednisolone 1 mg / kg per day for 1-3 days, followed by a decrease in dose in
  5. CHRONIC EOSINOPHILE PNEUMONIA
    It differs from the Loeffler syndrome with a longer (more than 4 weeks) and severe course until severe intoxication, fever, weight loss, the appearance of pleural effusion with a high content of eosinophils (Lera-Kindberg syndrome). The prolonged course of pulmonary eosinophilia, as a rule, is the result of an inadequate examination of the patient in order to determine its cause. In addition to the reasons,
  6. PULMONARY EOSINOPHILIA WITH ASTHMATIC SYNDROME
    This group of diseases can be attributed to bronchial asthma and diseases with a leading bronchial asthma syndrome, which are based on other etiological factors. These diseases include: 1. Allergic bronchopulmonary aspergillosis. 2. Tropical pulmonary eosinophilia. 3. Pulmonary eosinophilia with systemic manifestations. 4. Hypereosinophilic
  7. LITERATURE
    1. Diseases of the respiratory system: A guide for physicians: In 4 volumes. Ed. N.R.Paleeva. T.4. - M .: Medicine. - 1990. - P.22-39. 2. Silvestov VP, Bakulin MP Allergic lesions of the lungs // Klin.med. - 1987. - № 12. - P.117-122. 3. Exogenous allergic alveolitis / Ed. AG Khomenko, S. Mueller, V. Shilling. - M.: Medicine, 1987. -
  8. BRONCHOECTATIC DISEASE
    Bronchoectatic disease is an acquired (in some cases congenital) disease characterized by chronic suppuration in irreversibly altered (expanded, deformed) and functionally inferior bronchi predominantly in the lower parts of the lungs. ETIOLOGY AND PATHOGENESIS. Bronchiectasis is congenital in 6% of cases, being a defect of intrauterine development, the consequence
  9. CLASSIFICATION OF BRONCHOECTAZES
    (AI Borokhov, N.R.Paleev, 1990) 1. By origin: 1.1. Primary (congenital cysts) bronchiectasis. 1.1.1. Single (solitary). 1.1.2. Multiple. l..l-Z. Cystic lung. 1.2. Secondary (acquired) bronchiectasis. 2. By the form of bronchial dilatation: 2.1. Cylindrical. 2.2. Sacred. 2.3. Spindle-shaped. 2.4. Mixed. 3. The severity of the flow
  10. LITERATURE
    1. Diseases of the respiratory system. Manual for doctors edited by. N.R.Paleeva. - М .: Medicine, 1990. - Т.З, Т.4. 2. Okorokov A.N. Treatment of diseases of internal organs: Prakt.ru. In Зт. TI - Mn.Vysh.sh., Belmedkniga, 1997. 3. Harrison T.R. Internal illnesses. - M .: Medicine, T.7,
  11. REACHER'S DISEASE (SYNDROME)
    Reiter's disease (Reiter's syndrome, Fissinger-Leroy syndrome, urethro-oculo-synovial syndrome) is an inflammatory process developing in most cases in close chronological connection with infections of the genitourinary tract or intestine and manifested by the classic triad - urethritis, conjunctivitis, arthritis. The most often sick are young (20-40) men who have undergone urethritis. Women, children and the elderly
  12. DISEASE (SYNDROME) SCHEGREN
    The combination of dry keratoconjunctivitis, xerostomia and chronic polyarthritis was described in such detail by the Swedish ophthalmologist Shegren (Szegren, 1933), which soon attracted the attention of clinicians from different countries to this very peculiar clinical phenomenon, although single observations of such a triad or individual manifestations of secretory glandular failure were described earlier. Behind
  13. BRONCHIAL ASTHMA.
    The last decade is characterized by an increase in the incidence and severity of asthma (BA). On social significance, this condition is surely one of the first places among respiratory diseases. According to DJ Lane (1979), bronchial asthma is a disease that is relatively easy to recognize, but difficult to define. From the vast array of definitions,
  14. ETIOLOGY
    - One of the important etiological factors is heredity. Weighed heredity in bronchial asthma is found in 50-80% of patients. This is particularly evident in children: BA in one parent almost doubles the risk of developing the disease in the child, and asthma in both parents almost does not leave the child a chance to stay healthy. Numerous studies
  15. PATHOGENESIS
    Bronchial asthma is multifaceted and complex, it can not be considered one-sided, as a simple chain of pathological processes. Until now, there is no unified theory of pathogenesis. The development of this disease is based on complex immunological, non-immunological and neurohumoral mechanisms that are closely interrelated and interact with each other, cause hyperreactivity of the bronchial wall "
  16. CLINICAL PICTURE OF BRONCHIAL ASTHMA
    The main clinical sign of bronchial asthma is an attack of expiratory dyspnea due to reversible generalized airway obstruction as a result of bronchospasm, edema of the bronchial mucosa and hypersecretion of bronchial mucus. In the development of an attack of suffocation it is customary to distinguish three periods: I. The period of precursors or the prodromal period is characterized by the appearance
  17. TREATMENT OF BRONCHIAL ASTHMA
    There are no established treatment regimens for asthma. You can only talk about the principles of therapy for this contingent of patients, highlighting the principle of an individual approach to treatment. The most simple and effective method is etiotropic treatment, which consists in eliminating contact with the detected allergen. With increased sensitivity to home allergens or professional
  18. Diseases of Joints
    DISEASES
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