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Pulmonary adenomatosis

Chronic, long-lasting viral disease of sheep, cattle, horses and dogs, characterized by progressive metaplasia of the bronchial and alveolar epithelium, proliferation of glandular-like tumors in the lungs, becoming malignant. Adenomatosis of the lungs and a person gets sick.

The causative agent is a filterable virus.

Pre-slaughter diagnosis. Clinically, the disease manifests itself in the last stages and is accompanied by severe shortness of breath, which is especially noticeable during the driving of animals.

Post-mortem diagnosis. The mucous membrane of the larynx and trachea is edematous and cyanotic. In the lungs, there are limited or diffuse foci of dense consistency, grayish-white or grayish-pink. From the surface, the lesions are as if slightly sunken. When the lungs are cut from a small and large bronchi, a clear foamy liquid flows down, sometimes in the form of viscous mucus. Bronchial and mediastinal lymph nodes are enlarged, dense to the touch, with a turbid lymph from the surface of their incision.

Differential diagnosis.
With the differentiation of adenomatosis and diseases similar to it, it is necessary to exclude dictomaculosis vermin pneumonia, atelectasis of the lungs (more often the apex) resulting from lung inactivity during the postnatal period, catarrhal and other pneumonia resulting from a secondary infection (ornithosis, salmonellosis, chronic pasteurellosis ).

Veterinary-sanitary assessment. When establishing adenomatosis of the lungs, the release of meat and other products in its raw form is prohibited.

The carcasses of depleted animals, together with the heads and internal organs, are sent to the production of meat and bone meal, which is then fed only to the bird.

Carcasses and internal organs without visible pathological changes, obtained from the slaughter of sick and animals that are seropositive to the virus, are sent to cooked and cooked smoked sausages, meat breads or canned goods.

The head, bones, lungs and other pathologically changed organs, as well as the gastrointestinal tract, are sent for disposal. The skin and wool are disinfected.
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Pulmonary adenomatosis

  1. ADENOMATOSIS SHEEP AND GOATS
    Adenomatosis (Latin, English - Adenomatosis, Jaagsiekte; adenomatous bronchopneumonia, pulmonary adenomatosis) is a slowly progressing disease of sheep and goats, manifested by the development of glandular tissue (tumors) of the adenoma type in the lungs. Historical background, distribution, degree of danger and damage. The disease is first mentioned as a major epizootic in South Africa in the second
  2. Pulmonary sheep adenomatosis
    Adenomatosis of the lungs of sheep (adenomatosis ovium) is a chronic disease that is characterized by a long incubation period and progressive lesion of the lungs with the proliferation of gland-like lesions in them. Etiology. The disease is caused by an RNA-containing virus belonging to the family Retroviridae, the genus Lentivirus. Epizootological data. The disease is recorded in many
  3. LUNG DISEASES. CHRONIC DIFFUSE ASTHMA. INTERSTICIAL LUNG DISEASES. CANCER INFLAMMATORY DISEASES OF THE LUNGS. BRONCHIAL LUNG
    LUNG DISEASES. CHRONIC DIFFUSE ASTHMA. INTERSTICIAL LUNG DISEASES. CANCER INFLAMMATORY DISEASES OF THE LUNGS. BRONCHIAL
  4. Chronic diffuse inflammatory lung disease. Bronchial asthma. Lungs' cancer. Pneumoconiosis
    1. Main types of diffuse pulmonary lesions 1. interstitial 4. small-focal 2. obstructive 5. panacinal 3. restrictive 2. Causes of death in obstructive pulmonary emphysema 1. gas acidosis and coma 2. renal failure 3. left ventricular heart failure 4. right ventricular heart failure 5. lung collapse in spontaneous pneumothorax 3. Most important
  5. The mode of traditional artificial ventilation of the lungs with periodic inflation of the lungs
    It is well known that monotonous respiratory volume during mechanical ventilation increases the uneven ventilation of the lungs and contributes to their atelectasis. After all, even with independent breathing, a healthy person never breathes with the same breathing volume, the latter is constantly changing. In addition, a healthy person periodically makes "sighs" of increased volume and duration. To overcome
  6. Artificial ventilation of the lungs during operations on the lungs and mediastinal organs
    Single lung ventilation. A necessary condition for operations on the lungs — turning off the operated lung from ventilation in absolute terms (wet lung, pulmonary hemorrhage, unpressurized lung) or relative indications — puts the body in an unphysiological condition of functioning, leading to disturbances in gas exchange and blood circulation. However, the technique widely used in thoracic surgery
  7. Acute left ventricular failure - interstitial and alveolar pulmonary edema. Non-cardiogenic pulmonary edema.
    Pulmonary edema is cardiogenic and non-cardiogenic and is considered to be the immediate cause of death in every fourth deceased. Pathogenesis. In a healthy person, the hydrostatic pressure in the pulmonary capillaries is 7–9 mm Hg. st., it is slightly higher than that in the interstitium. The fluid is retained in the capillaries due to its viscous properties, rather high oncotic numbers.
  8. EXTERNAL RESPIRATION AND FUNCTIONS OF THE LUNG RESPIRATORY FUNCTION OF THE LUNGS AND PATHOPHYSIOLOGICAL MECHANISMS OF HYPOXEMIA AND HYPROCHEMISTRY
    The main function of the lungs - the exchange of oxygen and carbon dioxide between the external environment and the body - is achieved by a combination of ventilation, pulmonary circulation and diffusion of gases. Acute violations of one, two or all of these mechanisms lead to acute changes in gas exchange. Pulmonary ventilation. Indicators of pulmonary ventilation include tidal volume (Vт), respiratory rate (f), and minute volume
  9. CHRONIC OBSTRUCTIVE LUNG DISEASES / CHRONIC BRONCHITIS AND LUNG EMPHYSIS /
    Chronic obstructive pulmonary disease is a pathological condition characterized by the formation of chronic obstruction of the airways due to chronic bronchitis (CB) and / or pulmonary emphysema / EL /. Chronic obstructive pulmonary diseases are widespread. It is estimated that HB affects about 14–20% of the male and about 3–8% of the female adult population, but only
  10. Pulmonary edema caused by pulmonary edema, damage to the lungs and "volumatal injury" (volume trauma)
    Pathogenesis Even in the absence of an alveolar rupture, the use of excessive regional volumes undoubtedly damages the alveoli, regardless of whether the introduction of such volumes is caused by positive or negative pressure. Patients with acute respiratory distress syndrome appear to be at the highest risk: the prevalence of barotrauma in these conditions may exceed 50%. Have
  11. Correction methods for acute respiratory failure in acute lung injury / acute respiratory distress syndrome with a proven effect on mortality and ventilator-induced lung damage
    • ???? Ventilation with small respiratory volumes. The use of small respiratory volumes allows to reduce the manifestations of volutrauma and to avoid high transpulmonary pressures. According to the largest multicenter randomized controlled study conducted by ARDSnet in 41 centers and involving 861 patients, the use of small tidal volumes (6 ml / kg body weight) leads to
  12. 89. LUNG DISEASES CAUSED BY THE ACTION OF THE DUST FACTOR AND THE ACTION OF CHEMICAL FACTORS. PATHOGENESIS, CLINIC, TREATMENT PRINCIPLES AND PREVENTION. CLINIC OF TOXIC PULMONARY SWEAT, PRINCIPLES OF TREATMENT
    Pneumoconiosis is the development of fibrotic changes in the lungs as a result of prolonged inhalation. dust. According to etiology, 6 grams-PCs are distinguished: 1) silicosis-PC from inhalation of quartz dust (silica) 2) silicosis-PC from inhalation of silicate dust, silicon dioxide in a bound state with other elements #stostosis, talcosis. 3) metalloconioses - PC from dust Me: Al –aluminosis, iron oxides - siderosis. four)
  13. Barotrauma of the lungs
    Barotrauma during mechanical ventilation - damage to the lungs caused by the action of increased pressure in the respiratory tract. Two main mechanisms causing barotrauma should be indicated: 1) lung over-inflation; 2) uneven ventilation against the background of a modified lung structure. With barotrauma, air can enter the interstitium, mediastinum, neck tissue, cause pleural rupture, and even penetrate the abdominal cavity.
  14. LUNG BAROTRAVMA
    Barotrauma during mechanical ventilation - damage to the lungs caused by the action of increased pressure in the respiratory tract. Two main mechanisms causing barotrauma should be indicated: 1) lung over-inflation; 2) uneven ventilation against the background of a modified lung structure. With barotrauma, air can enter the interstitium, mediastinum, neck tissue, cause pleural rupture, and even penetrate the abdominal cavity.
  15. Lung features
    The lungs in children, as in adults, are divided into shares, shares into segments. The lungs have a lobular structure, the segments in the lungs are separated from each other by narrow grooves and septa of connective tissue. The main structural unit is the alveoli. Their number in a newborn is 3 times less than in an adult. Alveoli begin to develop from 4-6 weeks of age, their formation
  16. Restrictive lung diseases
    Causes of restrictive pulmonary pathology Causes of acute restrictive pathology: • ????? pulmonary edema; • ???? ARDS; •????aspiration; • ???? neurogenic edema; • ????? opioid overdose; • ???? congestive myocardial insufficiency; • ???? pleural effusion; •????pneumothorax; • ???? increase mediastinum; • ???? pneumomediastinum. Chronic lung disease leading to restrictive
  17. Vascular pathology of the lungs.
    Vascular pathology of the lungs occurs in various diseases of the lungs, heart and blood vessels, liver, and is described by various syndromes. The most important variants of lung vascular pathology are represented by the following groups of diseases: pulmonary edema; adult respiratory distress syndrome; pulmonary thromboembolism syndrome; syndromes of primary and secondary pulmonary hypertension;
  18. Restrictive lung diseases
    Restrictive diseases are characterized by a decrease in lung compliance. Lung volumes are below normal, while the volumetric flow rate on the exhalation is not reduced. Thus, FEV1 and FZHEL are reduced, but the value of the OFVch / FZHEL ratio remains normal. Restrictive diseases include many acute and chronic pathological conditions of the lungs, as well as lesions of the pleura, chest wall, diaphragm and
  19. Atelectasis of the lungs.
    Atelectasis is a pathological process characterized by a reversible incomplete unfolding or collapse of a part of the lung that previously contained air. The following types of atelectasis are distinguished: 1. Resorption (obstructive) atelectasis, which occurs with the complete obstruction of the bronchial tree with obstructive pulmonary diseases, aspiration of a foreign body, tumor growth. 2. Compression atelectasis
  20. Pulmonary edema
    Pulmonary edema is a pathological increase in the volume of extravascular fluid in the lungs. The main role is played by an increase in hydrostatic pressure in the pulmonary vessels, a decrease in plasma CODE, an increase in the permeability of the vascular wall. In 1896, E.G. Starling substantiated the theory of vascular resorption of fluid from connective tissue spaces into small vessels, according to which Qr = K (deltaR -
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