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Echinococcosis (echinococcosis) is a helminthic disease of many animals and humans, caused by the cestode larva Echinococcus granulosus fam. Taeniidae. The puberty cestode parasitizes in the intestines of dogs, wolves and other carnivores. Pigs, sheep, goats, cattle and other animals suffer from larval echinococcosis. Sick larval echinococcosis and man.
Etiology. Echinococcus granulosus larvae is a bubble that reaches the size of a pea to the head of a child. Outside the bubble is covered with a dense shell formed by the surrounding tissue of the host. The inner shell is germinative (germinal). On its surface, brood capsules and proto-scolexes are produced. Often, in addition to brood capsules and proto-scolexes, daughter, grand-daughter and grand-grandchild bubbles are produced from the germinal membrane, floating freely in the liquid.
In the bladder, as well as in the daughter, granddaughters and great-grandchildren, there is a light yellow, slightly opalescent liquid, in which brood capsules that contain proto-scolexes float.
Echinococci are localized more often in the liver, lungs, kidneys, but can also affect other organs.
The mature cestode (Echinococcus granulosus) is 3–6 mm long and consists of a scolex armed with 28–40 hooks and 3–4 segments. Localized in the small intestine of carnivores.
Epizootology. Infection of animals occurs through food and water contaminated with the eggs of an echinococcus. The main distributors of invasion among farm animals are dogs. The first segments with the feces of dogs are distinguished 1.5-2 months after invasion.
Symptoms and course depend on the localization of echinococcal bubbles. With liver damage, the disease is chronic. In the first days after infection, there is a lack of appetite, increased peristalsis, diarrhea, increased excitability, an increase in body temperature (by 0.5-1 ° C above normal).
Then signs of malaise gradually increase, the animals lie more, they avoid sharp movements. With a long course of the disease, a sharp emaciation, an increase in the volume of the abdomen due to an increase in the size of the liver or an edema of the abdominal cavity, soreness with pressure on the abdominal wall, jaundice or, conversely, anemical mucous membranes are observed. Sheep often have wool, cows have miscarriages, a decrease in milk yield.
Diagnosis. An intravital diagnosis in case of a larval echinococcosis in intermediate hosts can be made by X-ray and X-ray, an intradermal allergy test (native liquid taken sterile from an echinococcal bladder), serological reactions: RSHP, RNGA, RIF, etc. Posthumous diagnosis of echinococcosis, RSHP, RNGA, RIF, etc. Posthumous diagnosis of echinococcosis, RSHP, RNGA, RIF, etc. Posthumous diagnosis of echinococcosis, RSHP, RNGA, RIF, etc. opening of parasites in places of their localization does not cause special difficulties.
Treatment of animals with larval echinococcosis has not yet been developed. There is evidence that mebendazole is effective in echinococcosis of sheep (0.2 g / kg three times with an interval of 48 hours).
Prevention and control measures. For the prevention of echinococcosis, it is necessary not to allow dogs on the territory of slaughterhouses, to the places of opening and burial of animal carcasses. Prohibit feeding dogs uncleared waste slaughterhouses and household slaughter animals. All dogs should be subject to mandatory prophylactic deworming once a quarter. Hunting dogs must be dewormed every 1.5 months during the entire hunting season. Service dogs should be dewormed from December to April every 2 months, from May to November - after 45 days.
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Etiology. The causative agent of the hydatized echinococcosis is the larval stage of the small cestode, which has a scolex with 4 suckers and hooks and 3-4 proglotids filled with eggs. The larva is a single-chamber bladder whose wall consists of two layers of cells, external and internal, which form small parietal protrusions. The cavity of the bubble is filled with fluid. Echinococcus eggs highly resistant in external
- Echinococcosis of the brain
Echinococcosis is a parasitic disease that rarely affects the nervous system. Infection occurs when a human echinococcus eggs enter the human gastrointestinal tract - the larval stage of a small tapeworm (Taenia echinococcus) living in the intestines of dogs and wolves. Pathomorphology. There are two main forms of echinococcus - solitary and racemic. In the first case, there are single
The causative agent - Echinococcus granulosus - hydatious, Echinococcus multilocularis - alveolar, boa - in the larval stage. The path of transmission is contact life, food (from the coat of infection) Pathogenesis. Eggs get into the stomach - oncospheres - through the quit wall into the blood - into the liver, then they can get into the lungs, brain, and others. A bubble grows within 5-6 months, causing compression of the surrounding tissues - with-we volumetric
Etiology. The disease is even more rare than cysticercosis. Echinococcus - the young stage (Finn) of a small tapeworm - Taenia echinococcus, living in the intestines of dogs and wolves. Human infection occurs directly from dogs or due to the consumption of infected vegetables. In the stomach get eggs, which here lose their shell. The embryos released through the mucous membrane
- Helminthiasis (neurocysticercosis, echinococcosis, trichinosis)
Helminthiasis (neurocysticercosis, echinococcosis,
Echinococcosis. Zoonotic natural-anthropurgic helminthiasis, manifested in humans in two clinical forms - single-chamber (E. Granulosus) and alveolar (multi-chamber) (E. Multilocularis). The development takes place with the participation of two hosts - intermediate and final. Reservoir and sources: the final owner of the dog, in between animals. The transmission mechanism is fecal-oral,
- Differential diagnostics
The above tissue helminth infections should be differentiated between themselves, and in some cases (toxocarosis, trichinosis) - with the acute phase of other helminth infections (ascariasis, schistosomiasis, opisthorchiasis) and diseases, including those of an allergic nature, accompanied by high eosinophilia. Toxocarosis depending on the prevailing organ changes differentiate with asthma,
- Crib. Infectious diseases, 2010
Typhoid fever. Paratypus A and V. Holera. Amebiasis. Yersinioza. Ascariasis. Trichinosis. Typhus typhus, Brill-Zinsser b.n. Echinococcosis. Enterobiasis. Ku-fever. Lyme-borreliosis. Leptospirosis. Tularemia. Siberian ulcer. Meningococcal infection. Chuma. Column. Rabies. Ornithosis. Legionellosis. Rozh. Gripp. Food
- Dispensary observation
Patients with tissue helminth infections are subject to follow-up by an infectious diseases specialist at the place of residence. Patients operated on for echinococcosis and cysticercosis are observed by a surgeon, a neurosurgeon or an ophthalmologist at the site of the operation. When toxocarosis after the completion of etiopathogenetic therapy, control clinical blood tests and examination by ELISA with antigens are performed.
- ANIMAL INVASIVE DISEASES MEETING IN HUMAN, BUT NOT TRANSMITTED TO HIM THROUGH Slaughter Products
Echinococcosis Chronic disease of all types of agricultural and wild animals, as well as humans, caused by the larval stage of cestodes Echinococcus granulosus. Echinococcosis is often registered in Bashkortostan, Tatarstan, Krasnodar, Stavropol, Altai, Krasnoyarsk and Khabarovsk territories, Volgograd, Samara, Rostov, Orenburg, Magadan, Amur
- Treatment of tissue helminth infections
Treatment of patients with tissue helminth infections, regardless of the form of severity, is carried out in the hospital. In the acute period of the disease in severe forms of trichinosis, toxocariasis, cysticercosis, complicated forms of echinococcosis (rupture or suppuration of echinococcal cyst), bed rest is prescribed. Shown a full diet enriched with vitamins. Etiotropic therapy. Shown in all forms of tissue
Alveococcus multilocularis, - biohelminth, causative agent of alveococcosis. The disease foci are registered in Europe, Asia, and North America. MORPHOLOGICAL CHARACTERISTICS The tape forms of echinococcus and alveococcus are similar. They are distinguished by the structure of the uterus in mature proglottids: in alveococcus, the uterus is spherical, and in echinococcus - with lateral processes. Finna Alveococca is filled with gelatinous mass and