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IRIT AND IRID CYCLES

Iritis is inflammation of the iris, and iridocyclitis is inflammation of the iris and ciliary body. As a rule, iridocyclitis (uveitis) is observed in cats. The disease occurs as a result of injuries, viral, bacterial or toxoplasma infections, systemic fungal diseases, ascariasis, severe intoxication or metabolic disorders. In severe cases, iridocyclitis can lead to blindness.

Symptoms: lacrimation, photophobia, yellowness of the iris, narrowed pupil, poorly responsive to light, mucopurulent discharge from the inner corner of the eye.


First aid: transfer the cat to a darkened room and provide her peace. Instill eye drops with atropine, Iris eye drops, which have a pronounced bactericidal effect on most gram-positive and gram-negative bacteria (including Pseudomonas aeruginosa) in conjunctiva, cornea and deep endocular structures. Drops "Iris" are instilled in the conjunctival sac of 1-3 drops, depending on body weight. Gamavit intramuscularly.

Treatment will be prescribed by a veterinarian. Applying for qualified veterinary assistance is required.
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IRIT AND IRID CYCLES

  1. Differential diagnosis
    • Arthritis with rheumatism (rheumatic fever): - post-infection onset (catarrhal sore throat); - migratory lesion of large joints; - benign course of arthritis; - good clinical effect of salicylates, non-steroidal anti-inflammatory drugs; - a combination of arthritis with carditis, prolongation of symptoms of carditis after relief of arthritis. Elevated Captions
  2. Cardiac rheumatic fever in children
    Protocol code: 05-050а Profile: pediatric Stage: hospital Purpose of stage: 1. achievement of clinical and laboratory remission; 2. reduction (relief) of symptoms of circulatory failure; 3 hemodynamic stabilization, normalization of heart rate; 4. complete reverse development of laboratory activity indicators. Duration of treatment (days): 30 ICD code: 101.1 Acute rheumatic
  3. Uveitis
    A. Pathogenesis. The vascular membrane of the eye normally contains few T- and B-lymphocytes, mast cells and fibroblasts. When inflammation occurs, it is infiltrated by plasma cells, monocytes, T- and B-lymphocytes, some of these cells penetrate into aqueous humor. In the pathogenesis of uveitis, a soluble S-antigen with a molecular weight of 55,000 and retinal opsins plays an important role. B. History, physical and
  4. Periarteritis nodosa
    -general s-we: fever, weight loss, pallor -skin cm: subcutaneous nodules, erythematous rashes, petechiae, urticaria, necrosis, ulcers m: hypertension, heart attacks, bleeding, nephritis, pulmonary cm: vascular pneumonia, asthma, cavities, volatile infiltrates, heart attacks, pleurisy
  5. Rheumatoid arthritis
    - general s-we: sweating, pallor, weight loss -articular s-m: arthritis 3 months or more, effusion in the joint, joint contracture, morning stiffness, symmetrical lesion of small joints, tendosynovitis or bursitis - skin sm: rheumatoid nodules polymorphic rashes - cardiac SM: pericarditis, myocarditis, endocarditis, myocardial dystrophy, heart disease, aortitis - pulmonary SM: pulmonitis, pleurisy
  6. clinical classification, meningococcemia, treatment
    Meningococcal infection is an acute infectious disease caused by meningococcus (N. meningitidis), with an aerosol pathogen transmission mechanism; it is clinically characterized by damage to the mucous membrane of the nasopharynx (nasopharyngitis), generalization in the form of specific septicemia (meningococcemia) and inflammation of the pia mater (meningitis). Etiology: meningococcus - Gr-MB.
  7. Clinical options
    Postenterocolitic reactive arthritis is more often recorded during outbreaks of incidence of yersiniosis, salmonellosis, shigellez, helicobacteriosis, and clostridiosis. Enterocolitis is typically manifested by stool disorders, abdominal pain, usually in the right iliac region. The course of the disease is not severe, intestinal disorders stop in 1-2 weeks. even without using
  8. Test questions and tasks
    For questions 177–188, select one most correct answer. 177. With nodular periarteritis, the following symptoms are observed, with the exception of: A. Segmental or focal vascular lesions. B. Involvement of small and medium vessels in the process. B. Eosinophilia. G. The same stage of the pathological process in all affected vessels. D. Damage to vessels supplying the wall of large vessels.
  9. Syphilis
    The morphological element of the smallpox syphid (varicella syphilitica) is a superficial hemispherical pustule that appears against the background of a round red spot, 3–7 mm in diameter. The pustule is located in the center of the spot and is surrounded by a red rim. After a few days, the serous-purulent contents of the pustule thickens, its tire sinks in the center and dries into a thin crust.
  10. Visual impairment and visual pathway
    Cornea. The cornea, the main refracting surface of the eye, is highly sensitive to damaging environmental influences (direct trauma, drying, radiation and ionizing radiation), infectious agents (bacteria, viruses, especially herpes simplex and herpes zoster viruses, fungi, parasites), inflammatory processes, sometimes in combined with common skin diseases such as
  11. Vizer V.A .. Lectures on therapy, 2011
    On the subject - almost completely cover the difficulties in the course of hospital therapy, the issues of diagnosis, treatment, as stated, are concise and quite affordable. Allergic lung diseases Joint diseases Reiter's disease Sjogren's disease Bronchial asthma Bronchiectasis Hypertension Glomerulonephrosafasdit Esophageal hernia Destructive lung diseases
  12. ALLERGIC LUNG DISEASES
    In recent decades, a significant increase in the number. patients with allergic diseases of the bronchopulmonary apparatus. Allergic lung diseases include exogenous allergic alveolitis, pulmonary eosinophilia, and drug
  13. EXOGENOUS ALLERGIC ALVEOLITES
    Exogenous allergic alveolitis (synonym: hypersensitive pneumonitis, interstitial granulomatous alveolitis) is a group of diseases caused by intensive and, more rarely, prolonged inhalation of antigens of organic and inorganic dusts and are characterized by diffuse, in contrast to pulmonary eosinophilia, lesions of the alveolar and interstitial structures of the lungs. The emergence of this group
  14. TREATMENT
    1. General measures aimed at disconnecting the patient from the source of antigen: compliance with sanitary and hygienic requirements at the workplace, technological improvement of industrial and agricultural production, rational employment of patients. 2. Drug treatment. In the acute stage, prednisone 1 mg / kg per day for 1-3 days, followed by a dose reduction in
  15. Chronic Eosinophilic Pneumonia
    It differs from Leffler's syndrome in a longer (more than 4 weeks) and severe course up to severe intoxication, fever, weight loss, the appearance of pleural effusion with a high content of eosinophils (Lehr-Kindberg syndrome). The prolonged course of pulmonary eosinophilia, as a rule, is the result of under-examination of the patient in order to identify its cause. Besides the reasons
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