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66. CHRONIC GASTRITIS
Pain in the epigastric region is poorly expressed, is not clearly localized, does not affect the general condition of patients • Stomach dyspepsia: bursting in the epigastric region associated with eating; belching, nausea, vomiting, appetite disturbances • Dyspepsia: bloating, rumbling, flatulence, stool instability Asthenovegetative syndrome: weakness, increased fatigue, irritability and mood swings. • Chronic gastritis type A • Hypo- or achlorhydria • Signs of pernicious anemia • Concomitant thyroiditis. • Chronic gastritis type B • Pain in the epigastric region, shifted to the right subcostal region • In the later stages (after 10-20 years in 50% of patients) - signs of hypo- or achlorhydria. • Postgastrectomy gastritis • Violation of the production of Castle vasculature (Vitamin B12 deficiency) • Progression of changes to severe atrophy with the development of achlorhydria. Research methods • General blood test: pernicious anemia in chronic gastritis type A • Fecal analysis: possible presence of occult blood, as well as undigested food residues with low acidity • Biochemical analysis of blood: a decrease in total protein, inflammatory changes of varying severity (maximum with phlegmon gastric secretion) • Investigation of gastric secretion: a decrease or increase in the pH of fasting gastric juice with the use of specific irritants (cabbage broth, histamine), determination of pepsin content • PEGF with targeted biopsy: mucous membrane (edema, foci of hyperemia, petechiae, loosening and superficial ulceration, an increase in folds with hyperchlorhydria; pallor and atrophicity with hypo- or achlorhydria; free flow of bile into the stump of the acorns with reflux gastritis) • Radioscopy of the stomach: violations of the Historians, evacuation nations; required for differential diagnosis with diffuse gastric cancer • Identification of Helicobacter pylori.
Treatment • Diet. Depending on the type of secretion, 2 variants of therapeutic nutrition are distinguished: • In case of hypo- and achlorhydria in the acute phase, diet No. 1a is prescribed, then diet No. 2, after the end of the course of treatment, full nutrition • In hyperacid conditions, long-term diet No. 1. • Correction of disorders gastric secretion • With hypo- and achlorhydria - tincture of wormwood, infusion of dandelion root, natural gastric juice 1 tbsp. l half a glass of water in small sips while eating; polyenzyme preparations (festal, digestal, panzinorm-forte, mezim-fort) 1 tablet with a meal; chloride and chloride-bicarbonate sodium mineral waters • In case of hyperacid state - antacid, adsorbing and enveloping agents; histamine H2 receptor blockers (only with a combination of high hydrochloric acid production with erosion). • For gastritis caused by Helicobacter pylori, eradication (treatment course - 1-2 weeks) • Triple therapy • de-nol 120 mg 4 r / day for 28 days • metronidazole 200 mg 4 r / day for 10-14 days • tetracycline 500 mg 4 r / day for 10-14 days • Correction of disorders of the motor function of the stomach - myotropic antispasmodics (no-spa) • Treatment of concomitant diseases. • During the period of remission - spa treatment.
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66. CHRONIC GASTRITIS
- Chronic gastritis
CHRONIC GASTRY incremental functions of the stomach.
- Chronic gastritis (codes C 29.3 - 6)
Definition Chronic gastritis is a clinical morphological concept characterized by a violation of the physiological regeneration of the epithelium with an outcome in atrophy, impaired secretory function of the stomach, its motor and partly incretory activity. Statistics. Chronic gastritis is the most common digestive system disease, affecting about 50% of the adult population. In structure
- CHRONIC GASTRITIS
The most common disease of internal diseases. Chronic gastritis is a clinical anatomical concept. 1. Morphological changes in the mucous membrane are nonspecific, the inflammatory process is focal or diffuse. 2. Structural rearrangement of the mucosa with impaired regeneration and atrophy. 3. Nonspecific clinical manifestations. 4. Violations of the secretory, motor, partially incretory
- Chronic gastritis
- chronic inflammation of the gastric mucosa (diffuse or focal), accompanied by a violation of the physiological regeneration of the epithelium, its atrophy, functional insufficiency of the stomach, upset secretory, motor and often endocrine functions of the stomach The main clinical manifestations of chronic gastritis Local appearance of pain (dull, without radiation), severity and
- Chronic gastritis
Chronic gastritis is a long-running disease characterized by a chronic inflammatory process of the gastric mucosa and the gradual development of a number of its morphological changes (increase in its round-cell infiltration, impaired regeneration of the glandular epithelium with subsequent gradual atrophy of the epithelial cells). Judge now true
- Chronic gastritis and gastroduodenitis
Chronic gastritis (CG) is a chronic recurrent focal or diffuse inflammation of the gastric mucosa (submucosa) with a violation of physiological regeneration processes, with a tendency to progression, the development of atrophy, secretory insufficiency, which underlie digestive and metabolic disorders. Chronic gastroduodenitis (CGD) - chronic inflammation with structural
- Lectures. Chronic gastritis and gastroduodenitis, 2011
The lecture contains the latest information on the classification, etiology, clinic, pathogenesis, diagnosis and treatment of chronic gastritis and
- Nutrition for gastritis with low acidity (hypoacid gastritis)
With a decrease in acidity, that is, with insufficient secretion of gastric juice, clinical nutrition has the following goals: 1. Spare the diseased organ; 2. Stimulate the active production of gastric juice. In connection with a decrease in the amount of hydrochloric acid in the stomach, the excretion ability of the diseased organ with cooked food should be raised to improve protein digestion. Great importance
- Question 20 GASTRITIS
- inflammation of the gastric mucosa. There are acute and chronic gastritis. Acute gastritis is a polyetiological inflammation of the gastric mucosa caused by a number of irritants both from the outside and from the inside. Etiology and pathogenesis. Among exogenous factors, there are: nutritional errors in nutrition (quality and quantity of food eaten, especially plentiful food at night, eating
- Inflammation of the stomach (gastritis)
Distinguish between acute and chronic gastritis. Most often, gastritis occurs in children aged 5-6 years, 9-12 years, during periods of the most intensive development of all organs and systems. The incidence of girls and boys is the same, but during puberty is more common in girls. Acute gastritis is an acute inflammation of the gastric mucosa caused by short-term action of strong irritants.
Gastritis is an inflammation of the mucous membrane of the stomach, therefore see the article STOMACH (PROBLEMS), with the addition that a person with gastritis experiences or has experienced some kind of intense anger. See also explanation on page