home
about the project
Medical news
For authors
Licensed books on medicine
<< Previous Next >>

Tympanosclerosis. H-74.0

Treatment outcome: Clinical criteria for improving the patient's condition:

1. Normalization of laboratory parameters.

2. Improving the clinical symptoms of the disease (hearing loss).
<< Previous Next >>
= Skip to textbook content =

Tympanosclerosis. H-74.0

  1. Tympanosclerosis
    Tympanosclerosis belongs to the group of completed forms of fibrosing otitis media along with dry perforated otitis media and adhesive otitis media. From this point of view, tympanosclerosis can be considered as the outcome of catarrhal inflammation of the mucous membrane of the tympanic cavity (in the serous version). Tympanosclerosis is the result of a benign course of inflammation
  2. Vizer V.A .. Lectures on therapy, 2011
    On the subject - almost completely cover the difficulties in the course of hospital therapy, the issues of diagnosis, treatment, as stated, are concise and quite affordable. Allergic lung diseases Joint diseases Reiter's disease Sjogren's disease Bronchial asthma Bronchiectasis Hypertension Glomerulonephrosafasdit Esophageal hernia Destructive lung diseases
  3. ALLERGIC LUNG DISEASES
    In recent decades, a significant increase in the number. patients with allergic diseases of the bronchopulmonary apparatus. Allergic lung diseases include exogenous allergic alveolitis, pulmonary eosinophilia, and drug
  4. EXOGENOUS ALLERGIC ALVEOLITES
    Exogenous allergic alveolitis (synonym: hypersensitive pneumonitis, interstitial granulomatous alveolitis) is a group of diseases caused by intensive and, more rarely, prolonged inhalation of antigens of organic and inorganic dusts and are characterized by diffuse, in contrast to pulmonary eosinophilia, lesions of the alveolar and interstitial structures of the lungs. The emergence of this group
  5. TREATMENT
    1. General measures aimed at disconnecting the patient from the source of antigen: compliance with sanitary and hygienic requirements at the workplace, technological improvement of industrial and agricultural production, rational employment of patients. 2. Drug treatment. In the acute stage, prednisone 1 mg / kg per day for 1-3 days, followed by a dose reduction in
  6. Chronic Eosinophilic Pneumonia
    It differs from Leffler's syndrome in a longer (more than 4 weeks) and severe course up to severe intoxication, fever, weight loss, the appearance of pleural effusion with a high content of eosinophils (Lehr-Kindberg syndrome). The prolonged course of pulmonary eosinophilia, as a rule, is the result of an under-examination of the patient in order to identify its cause. Besides the reasons
  7. PULMONARY EOSINOPHILIA WITH ASTHMATIC SYNDROME
    This group of diseases can include bronchial asthma and diseases with a leading broncho-asthmatic syndrome, which are based on other etiological factors. These diseases include: 1. Allergic bronchopulmonary aspergillosis. 2. Tropical pulmonary eosinophilia. 3. Pulmonary eosinophilia with systemic manifestations. 4. Hyperoseosinophilic
  8. LITERATURE
    1. Respiratory diseases: A guide for doctors: In 4 volumes. Edited by N.R.Paleeva. T.4. - M .: Medicine. - 1990. - S. 22-39. 2. Silverstov V.P., Bakulin M.P. Allergic lung lesions // Klin.med. - 1987. - No. 12. - S.117-122. 3. Exogenous allergic alveolitis / Ed. A.G. Khomenko, St. Mueller, V. Schilling. - M.: Medicine, 1987. -
  9. BRONCHEECTATIC DISEASE
    Bronchiectatic disease is an acquired (in some cases congenital) disease characterized by a chronic suppurative process in irreversibly changed (enlarged, deformed) and functionally defective bronchi, mainly in the lower parts of the lungs. ETHIOLOGY AND PATHOGENESIS. Bronchiectasis are congenital in 6% of cases, as a fetal malformation, a consequence
  10. CLASSIFICATION OF BRONCHEKTASAS
    (A.I. Borokhov, N.R. Paleev, 1990) 1. By origin: 1.1. Primary (congenital cysts) bronchiectasis. 1.1.1. Single (solitary). 1.1.2. Multiple. l..l-Z. Cystic lung. 1.2. Secondary (acquired) bronchiectasis. 2. According to the form of bronchial enlargement: 2.1. Cylindrical. 2.2. Saccular. 2.3. Spindle-shaped. 2.4. Mixed. 3. The severity of the course
  11. LITERATURE
    1. Respiratory diseases. Manual for Doctors Edited. N.R.Paleeva. - M .: Medicine, 1990. - T.Z., T. 4. 2. Okorokov A.N. Treatment of diseases of internal organs: Practical guidance: In З. TI - Mn. Vysh.shk., Belmedkniga, 1997. 3. Harrison T.R. Internal illnesses. - M .: Medicine, T.7,
  12. DISEASE (SYNDROME) OF THE RATER
    Reiter’s disease (Reiter’s syndrome, Fissenge-Leroy’s syndrome, urethro-oculo-synovial syndrome) is an inflammatory process that develops in most cases in close chronological connection with infections of the genitourinary tract or intestines and manifested by the classical triad - urethritis, conjunctivitis, arthritis. Most often, young (20 - 40) men who have undergone urethritis are ill. Women, children and the elderly
  13. SHEHREN'S DISEASE (SYNDROME)
    The combination of dry keratoconjunctivitis, xerostomia, and chronic polyarthritis was described in such detail by the Swedish ophthalmologist Shegren (Shegren, 1933) that soon attracted the attention of clinicians from different countries to this very peculiar clinical phenomenon, although isolated observations of such a triad or individual manifestations of secretory glandular insufficiency were previously described. Behind
  14. BRONCHIAL ASTHMA.
    The last decade is characterized by an increase in the incidence and severity of bronchial asthma (BA). In terms of social significance, this condition confidently goes to one of the first places among respiratory diseases. According to DJ Lane (1979), bronchial asthma is a disease that is relatively easy to recognize but difficult to define. From a vast array of definitions,
  15. ETIOLOGY
    - One of the important etiological factors is heredity. Burdened heredity with bronchial asthma is found in 50-80% of patients. This is especially evident in children: AD in one of the parents almost doubles the risk of developing the disease in the child, and asthma in both parents almost leaves the child no chance to stay healthy. Numerous studies
  16. PATHOGENESIS
    Bronchial asthma is multifaceted and complex, it can not be considered one-sided, as a simple chain of pathological processes. There is still no unified theory of pathogenesis. The development of this disease is based on complex immunological, non-immunological and neurohumoral mechanisms that are closely related and interacting with each other, causing hyperresponsiveness of the bronchial wall "
  17. CLINICAL PICTURE OF BRONCHIAL ASTHMA
    The main clinical sign of bronchial asthma is an attack of expiratory dyspnea due to reversible generalized obstruction of the airways as a result of bronchospasm, edema of the bronchial mucosa and hypersecretion of bronchial mucus. In the development of an asthma attack, it is customary to distinguish three periods: I. The period of the precursors or the prodromal period is characterized by the appearance
  18. TREATMENT OF BRONCHIAL ASTHMA
    There are no established AD treatment regimens. We can only talk about the principles of therapy for this contingent of patients, highlighting the principle of an individual approach to treatment. The simplest and most effective method is etiotropic treatment, which consists in eliminating contact with the identified allergen. With hypersensitivity to home allergens or professional
Medical portal "MedguideBook" © 2014-2019
info@medicine-guidebook.com