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THERMAL AND CHEMICAL BURNS
Animals with thermal or chemical burns are killed for meat when they are in an incurable condition or their treatment and maintenance are not economically feasible in the future. Thermal burns in animals are the result of exposure to tissue of high temperature (fire, hot water, hot air), radiation energy, and electric current. As a result of thermal burns in animals, the skin and tissues located underneath it, the mucous membranes of the respiratory system (inga of hot air), the mucous membranes of the mouth, esophagus, and stomach (consumption of hot water, food) are affected.
Chemical burns with acids, alkalis, quicklime and other substances occur mainly due to the release of concentrated disinfectants and other solutions to the skin in the feed. In case of accidental consumption of solutions of caustic sodium, KI lot and some other caustic substances, instead of water, the animal has burns of the tissues of the oral cavity, esophagus, stomach.
Depending on the type of thermal or chemical agent, the intensity and duration of its action, the vitality of the affected tissue, the time elapsed after the burn, the fighting state of the animals can be very diverse. With minor burns of small areas of the skin, accompanied by hyperemia, swelling, slight soreness, the condition of the animals may be satisfactory. In severe cases (with extensive burns, charring, necrosis, tissue cleansing, complications with purulent processes), animals are depressed, their body temperature is elevated, and cardiac activity is impaired. These signs indicate severe pain irritation, intoxication of the body by tissue breakdown products, that is, the development of the so-called “burn disease” or even sepsis in them.
Post-mortem examination in animals with minor burns reveals mainly local tissue changes: hyperemia and swelling of the skin and subcutaneous tissue, slight swelling of the lymph nodes in the area of tissue disruption, enlargement and hyperemia of regional lymph nodes, congestive blood supply to the liver, kidneys, lungs, and hemorrhage in the kidneys.
In animals with extensive tissue damage, killed after 4-6 days. and more after a burn, the muscles in the place of the burn and around it are flabby, swollen, euro * pink, the lymph nodes are enlarged, swollen, with hemorrhages, the heart muscle is flabby, the spleen is sharply enlarged and blood-filled, the liver is enlarged, flabby, clay-gray or gray, in the kidneys heart attacks and hemorrhages. In case of complication of burns with purulent processes in animals, pleurisy, peritonitis, pneumonia, or a picture of septicemia are noted in addition to the above changes.
Sanitary assessment of slaughter products. If the burns are negligible, if the pre-slaughter condition of animals killed in the first 3 days. after a burn, satisfactory, damaged tissues are cut out and disposed of, and the carcass and organs are released without restrictions.
The use of products of slaughter of animals with lesions of a significant part, tissues or killed in the remote after a burn period depends on the results of bacteriological studies. Isolation of microflora from the liver, spleen, lymph nodes is considered as bacteremia or septic process, in such cases the meat and animal organs are disposed of.
Animals with significant burns and in serious condition are killed in the workshops using slaughter products for technical disposal or, after boiling, they are sent to feed the animals.
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THERMAL AND CHEMICAL BURNS
- Thermal and chemical burns
ICD-10 code T20 - T32 Diagnosis Diagnosis Mandatory Consciousness level, effectiveness and respiratory rate, blood pressure, heart rate, medical history, physical examination, burn surface area Consultation of a combiologist (traumatologist, surgeon) Laboratory tests: hemoglobin, blood gases, carboxyhemoglobin electrolytes (Na, K, Cl), coagulation indicators (APTT, PTV,
- Thermal and chemical burns of the respiratory tract
ICD-10 code T27 Diagnostics Diagnosis Mandatory Level of consciousness, effectiveness and respiratory rate, blood pressure, heart rate, medical history, physical examination R-graph of chest organs Bronchoscopy Laboratory tests: bacterial culture 2-3 days after a burn, hemoglobin, gases blood, carboxyhemoglobin, methemoglobin, electrolytes (Na, K, Cl), coagulation
- Thermal tracheal burns
Thermal burns of the trachea and other respiratory tract occur when inhaling the flame, hot air, smoke, steam. Purely thermal factors can be accompanied by chemical - toxic combustion products in smoke. A burn of the airways should be suspected in all cases when the damage was received indoors (fire in the house, basement, transport, mine, in the battle
- Thermal burns
Thermal burns - traumatic damage to the surface and deep tissues of the body under the influence of high temperature, accompanied by a general reaction with disruption of the activity of various organs and systems. Among the causes of death in childhood (up to 4 years), burns occupy the 2nd most frequent place (after road traffic injuries). Pathophysiology. Exposure to heat
- Thermal burns
Thermal burns are a type of injury that occurs as a result of exposure to body tissues of high temperature (flame, steam, boiling water, hot metals, gases, electromagnetic radiation of the optical range). Clinical diagnosis The severity of the condition of the affected child is determined by the depth and prevalence of tissue damage and manifestations of burn shock. Distinguish
- CHEMICAL BURNS
Chemical burns can be caused by organic and non-organic substances in the solid, liquid and gaseous state. The degree of damage depends on the amount of substance, its concentration and time of contact with the skin. The pathogenesis of chemical burns differs from the pathogenesis of thermal lesions. When concentrated acids act on the skin, tissue proteins coagulate (with the formation of
- Chemical burns of the esophagus
Esophageal burns are usually of a chemical nature, with the exception of the rarest cases of thermal burns. Chemical burns of the esophagus occur with the accidental or deliberate (with suicidal purpose) intake of aggressive fluids inside. Currently, the most common cause of such burns is acetic essence (80% solution of acetic acid). The pathogenesis of a chemical burn of the esophagus is quite typical.
- Chemical burns
Scope of the examination 1. Chemical burns cause corrosive liquids - concentrated acids and caustic alkalis. 2. Acids have a cauterizing and necrotic effect, coagulation of tissue proteins; alkalis dissolve proteins and cause kallikvatsionny necrosis. 3. Signs of a chemical burn if liquid gets inside are: burns on the face, lip mucosa, oropharynx, hoarseness
- Chemical burns of the respiratory and esophageal tract
Chemical burns of the respiratory tract Chemical burns occur as a result of ingestion or inhalation of concentrated chemical solutions (acids, alkalis, etc.). Most often, the vestibular part of the larynx is affected (epiglottis, scoop-epiglottis and vestibular folds, arytenoid cartilage). At the site of contact of the chemical agent with the mucous membrane, a local burn occurs
- CHEMICAL COMPOSITION OF A CELL AND ITS PHYSICAL AND CHEMICAL PROPERTIES
The elemental composition of the cell (protoplasm). To clearly imagine the biological and physicochemical properties of tissues, it is necessary to know the chemical composition of the protoplasm of the cell. In addition to water, there are a large number of elements in the protoplasm. The finest chemical studies have found that out of 104 elements of the periodic system of D. I. Mendeleev, protoplasm contains 96. Four
- Therapeutic tactics for thermal lesions
Thermal damage occurs as a result of exposure to thermal energy (burns) or low temperature (frostbite). Burns (burn disease) - tissue damage resulting from local thermal, chemical, electrical or radiation exposure. In depth, 4 degrees of damage are distinguished. I degree - erythema, swelling and pain of reddened skin is noted. Phenomena pass through 3-5